Zinc transporter Slc39a8 is essential for cardiac ventricular compaction.

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Zinc transporter Slc39a8 is essential for cardiac ventricular compaction.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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Zinc transporter Slc39a8 is essential for cardiac ventricular compaction.

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Zinc transporter Slc39a8 is essential for cardiac ventricular compaction.

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Zinc transporter Slc39a8 is essential for cardiac ventricular compaction.

@en

Zinc transporter Slc39a8 is essential for cardiac ventricular compaction.

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Zinc transporter Slc39a8 is essential for cardiac ventricular compaction.

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Zinc transporter Slc39a8 is essential for cardiac ventricular compaction.

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P1476

Zinc transporter Slc39a8 is essential for cardiac ventricular compaction.

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P2093

Daniel J Rader

http://www.w3.org/2001/XMLSchema#string

Deqiang Li

http://www.w3.org/2001/XMLSchema#string

Feiyan Liu

http://www.w3.org/2001/XMLSchema#string

Jonathan A Epstein

http://www.w3.org/2001/XMLSchema#string

Lan Cheng

http://www.w3.org/2001/XMLSchema#string

Li Li

http://www.w3.org/2001/XMLSchema#string

Nicholas J Hand

http://www.w3.org/2001/XMLSchema#string

Wen Lin

http://www.w3.org/2001/XMLSchema#string

P2860

Notch signaling is essential for ventricular chamber development

Mycobacterium bovis BCG cell wall and lipopolysaccharide induce a novel gene, BIGM103, encoding a 7-TM protein: identification of a new protein family having Zn-transporter and Zn-metalloprotease signatures

Disruption of hyaluronan synthase-2 abrogates normal cardiac morphogenesis and hyaluronan-mediated transformation of epithelium to mesenchyme

DAVID: Database for Annotation, Visualization, and Integrated Discovery

Mutations in sarcomere protein genes in left ventricular noncompaction

Contemporary definitions and classification of the cardiomyopathies: an American Heart Association Scientific Statement from the Council on Clinical Cardiology, Heart Failure and Transplantation Committee; Quality of Care and Outcomes Research and F

Novel gene mutations in patients with left ventricular noncompaction or Barth syndrome.

Left ventricular noncompaction cardiomyopathy: cardiac, neuromuscular, and genetic factors.

Counting the zinc-proteins encoded in the human genome.

Developmental patterning of the myocardium.

P304

826-833

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P31

scholarly article

P356

10.1172/JCI96993

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

128

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P577

2018-01-16T00:00:00Z

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P698

29337306

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P921

zinc II ion transmembrane import

Solute carrier family 39 member 8

Solute carrier family 39 (metal ion transporter), member 8

P932

5785267

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