Inhibition of Osteoblast Function by Brucella abortus is Reversed by Dehydroepiandrosterone and Involves ERK1/2 and Estrogen Receptor.

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Inhibition of Osteoblast Function by Brucella abortus is Reversed by Dehydroepiandrosterone and Involves ERK1/2 and Estrogen Receptor.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

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2018年學術文章

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2018年學術文章

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name

Inhibition of Osteoblast Funct ...... ERK1/2 and Estrogen Receptor.

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Inhibition of Osteoblast Funct ...... ERK1/2 and Estrogen Receptor.

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Item

label

Inhibition of Osteoblast Funct ...... ERK1/2 and Estrogen Receptor.

@en

Inhibition of Osteoblast Funct ...... ERK1/2 and Estrogen Receptor.

@nl

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Inhibition of Osteoblast Funct ...... ERK1/2 and Estrogen Receptor.

@en

Inhibition of Osteoblast Funct ...... ERK1/2 and Estrogen Receptor.

@nl

P1476

Inhibition of Osteoblast Funct ...... s ERK1/2 and Estrogen Receptor

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Andrea Elena Iglesias Molli

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Ayelén Ivana Pesce Viglietti

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Gloria Edith Cerrone

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Guillermo Hernán Giambartolomei

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María Virginia Gentilini

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Paula Constanza Arriola Benitez

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P2860

Endogenous glucocorticoids decrease skeletal angiogenesis, vascularity, hydration, and strength in aged mice.

The biology of the glucocorticoid receptor: new signaling mechanisms in health and disease

Cellular processing of the glucocorticoid receptor gene and protein: new mechanisms for generating tissue-specific actions of glucocorticoids.

Brucella intracellular life: from invasion to intracellular replication.

Prevention of glucocorticoid induced-apoptosis of osteoblasts and osteocytes by protecting against endoplasmic reticulum (ER) stress in vitro and in vivo in female mice.

Potential role of fibroblast-like synoviocytes in joint damage induced by Brucella abortus infection through production and induction of matrix metalloproteinases.

Glucocorticoid-induced apoptosis and glucocorticoid resistance: molecular mechanisms and clinical relevance.

Brucella abortus invasion of osteoblasts inhibits bone formation.

The paradox of matrix metalloproteinases in infectious disease.

Dehydroepiandrosterone stimulates endothelial proliferation and angiogenesis through extracellular signal-regulated kinase 1/2-mediated mechanisms

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scholarly article

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10.3389/FIMMU.2018.00088

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P478

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M. Victoria Delpino

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2018-01-26T00:00:00Z

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29434601

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osteoblast

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5790783

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