Inhibiting trophoblast PAR-1 overexpression suppresses sFlt-1-induced anti-angiogenesis and abnormal vascular remodeling: a possible therapeutic approach for preeclampsia.

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Inhibiting trophoblast PAR-1 overexpression suppresses sFlt-1-induced anti-angiogenesis and abnormal vascular remodeling: a possible therapeutic approach for preeclampsia.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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Inhibiting trophoblast PAR-1 o ...... tic approach for preeclampsia.

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Inhibiting trophoblast PAR-1 o ...... tic approach for preeclampsia.

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Inhibiting trophoblast PAR-1 o ...... tic approach for preeclampsia.

@en

Inhibiting trophoblast PAR-1 o ...... tic approach for preeclampsia.

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Inhibiting trophoblast PAR-1 o ...... tic approach for preeclampsia.

@en

Inhibiting trophoblast PAR-1 o ...... tic approach for preeclampsia.

@nl

P1476

Inhibiting trophoblast PAR-1 o ...... tic approach for preeclampsia.

@en

P2093

Di Wu

http://www.w3.org/2001/XMLSchema#string

Li Zou

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QingQing Luo

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XiaoPing Liu

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XiaoXia Liu

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YanFang Zheng

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Yin Zhao

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P2860

Excessive placental secretion of neurokinin B during the third trimester causes pre-eclampsia

Effects of thrombin, PAR-1 activating peptide and a PAR-1 antagonist on umbilical artery resistance in vitro

Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia

Pathogenesis and genetics of pre-eclampsia.

Pre-eclampsia.

Effects of netrin-1 and netrin-1 knockdown on human umbilical vein endothelial cells and angiogenesis of rat placenta.

Protease-activated receptor 1 and 4 signal inhibition reduces preterm neonatal hemorrhagic brain injury.

Thrombin regulates soluble fms-like tyrosine kinase-1 (sFlt-1) expression in first trimester decidua: implications for preeclampsia

Thrombin receptors and protease-activated receptor-2 in human placentation: receptor activation mediates extravillous trophoblast invasion in vitro.

Over-expression of the thrombin receptor (PAR-1) in the placenta in preeclampsia: a mechanism for the intersection of coagulation and inflammation.

P31

scholarly article

P356

10.1093/MOLEHR/GAX068

http://www.w3.org/2001/XMLSchema#string

P577

2018-01-09T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29325127

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P921

pre-eclampsia

overexpression