Decreased activity of plasma ADAMTS13 are related to enhanced cytokinemia and endotoxemia in patients with acute liver failure.

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Decreased activity of plasma ADAMTS13 are related to enhanced cytokinemia and endotoxemia in patients with acute liver failure.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年學術文章

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name

Decreased activity of plasma A ...... ents with acute liver failure.

@en

Decreased activity of plasma A ...... ents with acute liver failure.

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type

Item

label

Decreased activity of plasma A ...... ents with acute liver failure.

@en

Decreased activity of plasma A ...... ents with acute liver failure.

@nl

prefLabel

Decreased activity of plasma A ...... ents with acute liver failure.

@en

Decreased activity of plasma A ...... ents with acute liver failure.

@nl

P1476

Decreased activity of plasma A ...... ents with acute liver failure.

@en

P2093

Hideto Kawaratani

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Hiroaki Takaya

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Hiroshi Fukui

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Hitoshi Yoshiji

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Kazuya Sakai

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Masanori Matsumoto

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Yoshihiro Fujimura

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P2860

Structure of von Willebrand factor-cleaving protease (ADAMTS13), a metalloprotease involved in thrombotic thrombocytopenic purpura

Inflammatory cytokines inhibit ADAMTS13 synthesis in hepatic stellate cells and endothelial cells

Mutations in a member of the ADAMTS gene family cause thrombotic thrombocytopenic purpura.

Severe secondary deficiency of von Willebrand factor-cleaving protease (ADAMTS13) in patients with sepsis-induced disseminated intravascular coagulation: its correlation with development of renal failure.

Thrombotic thrombocytopenic purpura associated with pegylated-interferon alpha-2a by an ADAMTS13 inhibitor in a patient with chronic hepatitis C.

Comprehensive analysis of ADAMTS13 in patients with liver cirrhosis.

First symptoms in patients with thrombotic thrombocytopenic purpura: what are they and when do they occur?

von Willebrand factor-cleaving protease in thrombotic thrombocytopenic purpura and the hemolytic-uremic syndrome.

Involvement of large plasma von Willebrand factor (vWF) multimers and unusually large vWF forms derived from endothelial cells in shear stress-induced platelet aggregation.

Relation of endotoxin, endotoxin binding proteins and macrophages to severe alcoholic liver injury and multiple organ failure.

P304

277-285

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scholarly article

P356

10.3892/BR.2017.945

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P478

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P577

2017-07-19T00:00:00Z

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28894574

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P932

5582607

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