GnRH receptor expression and reproductive function depend on JUN in the GnRH receptor-expressing cells.

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GnRH receptor expression and reproductive function depend on JUN in the GnRH receptor-expressing cells.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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GnRH receptor expression and r ...... nRH receptor-expressing cells.

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GnRH receptor expression and r ...... nRH receptor-expressing cells.

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GnRH receptor expression and r ...... nRH receptor-expressing cells.

@en

GnRH receptor expression and r ...... nRH receptor-expressing cells.

@nl

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GnRH receptor expression and r ...... nRH receptor-expressing cells.

@en

GnRH receptor expression and r ...... nRH receptor-expressing cells.

@nl

P1476

GnRH Receptor Expression and R ...... GnRH Receptor‒Expressing Cells

@en

P2093

Carrie R Jonak

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Nancy M Lainez

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Ulrich Boehm

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P2860

Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects

Follicle-stimulating hormone synthesis and fertility depend on SMAD4 and FOXL2.

Luteinizing hormone deficiency and female infertility in mice lacking the transcription factor NGFI-A (Egr-1)

The gonadotropin releasing hormone (GnRH) receptor activating sequence (GRAS) is a composite regulatory element that interacts with multiple classes of transcription factors including Smads, AP-1 and a forkhead DNA binding protein

Optimized amplification and single-cell analysis identify GnRH-mediated activation of Rap1b in primary rat gonadotropes

c-Jun is essential for organization of the epidermal leading edge

AP-1 in cell proliferation and survival

AP-1 function and regulation

Dose-dependent switch in response of gonadotropin-releasing hormone (GnRH) neurons to GnRH mediated through the type I GnRH receptor.

Gonadotropin and kisspeptin gene expression, but not GnRH, are impaired in cFOS deficient mice.

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1496-1510

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scholarly article

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10.1210/EN.2017-00844

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English

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159

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D Coss

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2018-03-01T00:00:00Z

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29409045

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P932

5839737

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