Developmental differences in the responses of IL-6 and IL-13 transgenic mice exposed to hyperoxia. - Wikidata - awgldk - TriplyDB

Developmental differences in the responses of IL-6 and IL-13 transgenic mice exposed to hyperoxia.

Developmental differences in the responses of IL-6 and IL-13 transgenic mice exposed to hyperoxia.

http://www.wikidata.org/entity/Q50232144

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Understanding the Impact of Infection, Inflammation, and Their Persistence in the Pathogenesis of Bronchopulmonary Dysplasia Alpha-phenyl-N-tert-butylnitrone attenuates hyperoxia-induced lung injury by down-modulating inflammation in neonatal rats Postnatal inflammation in the pathogenesis of bronchopulmonary dysplasia Sustained hyperoxia-induced NF-κB activation improves survival and preserves lung development in neonatal mice.Foxp1/2/4-NuRD interactions regulate gene expression and epithelial injury response in the lung via regulation of interleukin-6 Dantrolene mitigates caerulein-induced pancreatitis in vivo in mice.Hyperoxia-derived lung damage in preterm infants.A potential role of the JNK pathway in hyperoxia-induced cell death, myofibroblast transdifferentiation and TGF-β1-mediated injury in the developing murine lung.Timing of umbilical cord blood derived mesenchymal stem cells transplantation determines therapeutic efficacy in the neonatal hyperoxic lung injury.A critical regulatory role for macrophage migration inhibitory factor in hyperoxia-induced injury in the developing murine lung.Conditional overexpression of TGFβ1 promotes pulmonary inflammation, apoptosis and mortality via TGFβR2 in the developing mouse lung.Targeting inflammation to prevent bronchopulmonary dysplasia: can new insights be translated into therapies?Immune response to intrapharyngeal LPS in neonatal and juvenile mice Differential Expression of the Activator Protein 1 Transcription Factor Regulates Interleukin-1ß Induction of Interleukin 6 in the Developing Enterocyte.Increased hyperoxia-induced lung injury in nitric oxide synthase 2 null mice is mediated via angiopoietin 2.Hyperoxia promotes polarization of the immune response in ovalbumin-induced airway inflammation, leading to a TH17 cell phenotype Heritability of bronchopulmonary dysplasia, defined according to the consensus statement of the national institutes of health.Long-term (postnatal day 70) outcome and safety of intratracheal transplantation of human umbilical cord blood-derived mesenchymal stem cells in neonatal hyperoxic lung injury Small molecular modulation of macrophage migration inhibitory factor in the hyperoxia-induced mouse model of bronchopulmonary dysplasia.Developmental regulation of NO-mediated VEGF-induced effects in the lung.The Fas system confers protection against alveolar disruption in hyperoxia-exposed newborn mice Hyperoxia and interferon-γ-induced injury in developing lungs occur via cyclooxygenase-2 and the endoplasmic reticulum stress-dependent pathway.Broad DNA repair responses in neural injury are associated with activation of the IL-6 pathway in cholesterol-fed rabbits.Nrf2 increases survival and attenuates alveolar growth inhibition in neonatal mice exposed to hyperoxia Sphingosine kinase 1 deficiency confers protection against hyperoxia-induced bronchopulmonary dysplasia in a murine model: role of S1P signaling and Nox proteins Age, strain, and gender as factors for increased sensitivity of the mouse lung to inhaled ozone Hyperoxia exacerbates postnatal inflammation-induced lung injury in neonatal BRP-39 null mutant mice promoting the M1 macrophage phenotype.Transcriptional responses of neonatal mouse lung to hyperoxia by Nrf2 status.Inhibition of Regulatory-Associated Protein of Mechanistic Target of Rapamycin Prevents Hyperoxia-Induced Lung Injury by Enhancing Autophagy and Reducing Apoptosis in Neonatal Mice.Hyperoxia-induced p47phox activation and ROS generation is mediated through S1P transporter Spns2, and S1P/S1P1&2 signaling axis in lung endothelium.A role for matrix metalloproteinase 9 in IFNγ-mediated injury in developing lungs: relevance to bronchopulmonary dysplasia.IFN-γ and IP-10 in tracheal aspirates from premature infants: relationship with bronchopulmonary dysplasia.Genetic ablation of Bach1 gene enhances recovery from hyperoxic lung injury in newborn mice via transient upregulation of inflammatory genes.Effects of hyperoxia on transdifferentiation of primary cultured typeII alveolar epithelial cells from premature rats.Human umbilical cord blood-derived mesenchymal stem cells attenuate hyperoxia-induced lung injury in neonatal rats.Early gestational mesenchymal stem cell secretome attenuates experimental bronchopulmonary dysplasia in part via exosome-associated factor TSG-6.

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Developmental differences in the responses of IL-6 and IL-13 transgenic mice exposed to hyperoxia.

http://www.wikidata.org/entity/Q50232144

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2007 nî lūn-bûn

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American Journal of Physiology - Lung Cellular and Molecular Physiology

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Jack A Elias

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Rayman Choo-Wing

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P2860

Excess circulating angiopoietin-2 may contribute to pulmonary vascular leak in sepsis in humans

Effects of early inhaled beclomethasone therapy on tracheal aspirate inflammatory mediators IL-8 and IL-1ra in ventilated preterm infants at risk for bronchopulmonary dysplasia.

Hyperoxia-induced apoptosis and Fas/FasL expression in lung epithelial cells.

Bcl-2-related protein A1 is an endogenous and cytokine-stimulated mediator of cytoprotection in hyperoxic acute lung injury.

Cytokines in tolerance to hyperoxia-induced injury in the developing and adult lung.

Developmental differences in the role of interleukins in hyperoxic lung injury in animal models.

Early increase of TNF alpha and IL-6 in tracheobronchial aspirate fluid indicator of subsequent chronic lung disease in preterm infants

Alveolar cell death in hyperoxia-induced lung injury.

Developmental aspects of experimental pulmonary oxygen toxicity.

Hyperoxia causes angiopoietin 2-mediated acute lung injury and necrotic cell death

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