IL-18 is induced and IL-18 receptor alpha plays a critical role in the pathogenesis of cigarette smoke-induced pulmonary emphysema and inflammation.
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The IL-1 cytokine family and its role in inflammation and fibrosis in the lungRole of the inflammasome-caspase1/11-IL-1/18 axis in cigarette smoke driven airway inflammation: an insight into the pathogenesis of COPDA Mitochondrial Perspective of Chronic Obstructive Pulmonary Disease PathogenesisGenetically manipulated mouse models of lung disease: potential and pitfallsEndothelial cell adhesion molecule CD146: implications for its role in the pathogenesis of COPDP2X7 receptor and caspase 1 activation are central to airway inflammation observed after exposure to tobacco smokeCytokine inhibition in the treatment of COPD.Cytotoxic potential of lung CD8(+) T cells increases with chronic obstructive pulmonary disease severity and with in vitro stimulation by IL-18 or IL-15.Identification of Novel Therapeutic Targets in COPD.Role for cathepsin K in emphysema in smoke-exposed guinea pigsExposure to cigarette smoke inhibits the pulmonary T-cell response to influenza virus and Mycobacterium tuberculosis.Differential expression and function of breast regression protein 39 (BRP-39) in murine models of subacute cigarette smoke exposure and allergic airway inflammation.Disruption of the transcription factor Nrf2 promotes pro-oxidative dendritic cells that stimulate Th2-like immunoresponsiveness upon activation by ambient particulate matter.Inflammasome Activity in Non-Microbial Lung Inflammation.Role of breast regression protein-39 in the pathogenesis of cigarette smoke-induced inflammation and emphysema.MicroRNA-21 limits in vivo immune response-mediated activation of the IL-12/IFN-gamma pathway, Th1 polarization, and the severity of delayed-type hypersensitivity.Suppression of NLRX1 in chronic obstructive pulmonary disease.IL-18 induces emphysema and airway and vascular remodeling via IFN-γ, IL-17A, and IL-13Role of Chitinase 3-Like-1 in Interleukin-18-Induced Pulmonary Type 1, Type 2, and Type 17 Inflammation; Alveolar Destruction; and Airway Fibrosis in the Murine Lung.Chitinase 3-like-1 promotes Streptococcus pneumoniae killing and augments host tolerance to lung antibacterial responses.Cigarette smoke selectively enhances viral PAMP- and virus-induced pulmonary innate immune and remodeling responses in mice.Tobacco smoke induced COPD/emphysema in the animal model-are we all on the same page?Role of IL-18 in second-hand smoke-induced emphysemaInflammation and immune response in COPD: where do we stand?Mechanisms of cigarette smoke-induced COPD: insights from animal models.Roles for proteinases in the pathogenesis of chronic obstructive pulmonary disease.Nonredundant functions of alphabeta and gammadelta T cells in acrolein-induced pulmonary pathology.Increased cytokine production in interleukin-18 receptor alpha-deficient cells is associated with dysregulation of suppressors of cytokine signaling.New insights into mechanisms controlling the NLRP3 inflammasome and its role in lung disease.The possible role of granzyme B in the pathogenesis of chronic obstructive pulmonary disease.Cigarette smoke destabilizes NLRP3 protein by promoting its ubiquitination.Inflammatory response of lung macrophages and epithelial cells to tobacco smoke: a literature review of ex vivo investigations.Inflammatory triggers associated with exacerbations of COPD orchestrate plasticity of group 2 innate lymphoid cells in the lungs.Current concepts on oxidative/carbonyl stress, inflammation and epigenetics in pathogenesis of chronic obstructive pulmonary disease.Genetics of interleukin 1 receptor-like 1 in immune and inflammatory diseases.Cigarette smoke and ozone effect on murine inflammatory responses.Mitochondrial Regulation of Inflammasome Activation in Chronic Obstructive Pulmonary Disease.Correlation of NLRP3 with severity and prognosis of coronary atherosclerosis in acute coronary syndrome patients.Quantification and evaluation of the role of antielastin autoantibodies in the emphysematous lungPulmonary epithelial neuropilin-1 deletion enhances development of cigarette smoke-induced emphysema.
P2860
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P2860
IL-18 is induced and IL-18 receptor alpha plays a critical role in the pathogenesis of cigarette smoke-induced pulmonary emphysema and inflammation.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
IL-18 is induced and IL-18 rec ...... ry emphysema and inflammation.
@en
IL-18 is induced and IL-18 rec ...... ry emphysema and inflammation.
@nl
type
label
IL-18 is induced and IL-18 rec ...... ry emphysema and inflammation.
@en
IL-18 is induced and IL-18 rec ...... ry emphysema and inflammation.
@nl
prefLabel
IL-18 is induced and IL-18 rec ...... ry emphysema and inflammation.
@en
IL-18 is induced and IL-18 rec ...... ry emphysema and inflammation.
@nl
P2093
P1476
IL-18 is induced and IL-18 rec ...... ry emphysema and inflammation.
@en
P2093
Carolyn Rochester
Geoffrey Chupp
Hilary Cain
Ho Joo Yoon
Jack A Elias
Min-Jong Kang
Robert J Homer
Soo Jung Cho
P304
P356
10.4049/JIMMUNOL.178.3.1948
P407
P577
2007-02-01T00:00:00Z