Rap1/B-Raf signaling is activated in neuroendocrine tumors of the digestive tract and Raf kinase inhibition constitutes a putative therapeutic target.

Rap1/B-Raf signaling is activated in neuroendocrine tumors of the digestive tract and Raf kinase inhibition constitutes a putative therapeutic target.

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http://www.wikidata.org/entity/Q50471116
From targets to treatments: a review of molecular targets in pancreatic neuroendocrine tumorsPI3K-AKT-mTOR-signaling and beyond: the complex network in gastroenteropancreatic neuroendocrine neoplasmsDown-Regulation of miR-129-5p and the let-7 Family in Neuroendocrine Tumors and Metastases Leads to Up-Regulation of Their Targets Egr1, G3bp1, Hmga2 and Bach1.ERK/pERK expression and B-raf mutations in colon adenocarcinomas: correlation with clinicopathological characteristics.Protein kinase Cδ inactivation inhibits cellular proliferation and decreases survival in human neuroendocrine tumors.Decoding the Molecular and Mutational Ambiguities of Gastroenteropancreatic Neuroendocrine Neoplasm Pathobiology.The role of cell lines in the study of neuroendocrine tumors.EGFR/TGFα and TGFβ/CTGF Signaling in Neuroendocrine Neoplasia: Theoretical Therapeutic Targets.Anti-proliferative and anti-secretory effects of everolimus on human pancreatic neuroendocrine tumors primary cultures: is there any benefit from combination with somatostatin analogs?The clinical implications and biologic relevance of neurofilament expression in gastroenteropancreatic neuroendocrine neoplasms.Pancreatic endocrine tumors: a report on a patient treated with sorafenib.Treatment of gastrointestinal neuroendocrine tumors with inhibitors of growth factor receptors and their signaling pathways: recent advances and future perspectives.Measurement of circulating transcripts and gene cluster analysis predicts and defines therapeutic efficacy of peptide receptor radionuclide therapy (PRRT) in neuroendocrine tumors.Phosphorylation of Rap1 by cAMP-dependent Protein Kinase (PKA) Creates a Binding Site for KSR to Sustain ERK Activation by cAMP.
P2860

Rap1/B-Raf signaling is activated in neuroendocrine tumors of the digestive tract and Raf kinase inhibition constitutes a putative therapeutic target.

Item
http://www.wikidata.org/entity/Q50471116
description
2007 nî lūn-bûn
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2007年の論文
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2007年学术文章
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name
Rap1/B-Raf signaling is activa ...... a putative therapeutic target.
@en
Rap1/B-Raf signaling is activa ...... a putative therapeutic target.
@nl
type
label
Rap1/B-Raf signaling is activa ...... a putative therapeutic target.
@en
Rap1/B-Raf signaling is activa ...... a putative therapeutic target.
@nl
prefLabel
Rap1/B-Raf signaling is activa ...... a putative therapeutic target.
@en
Rap1/B-Raf signaling is activa ...... a putative therapeutic target.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P577
P698
P356
P1433
P1476
Rap1/B-Raf signaling is activa ...... a putative therapeutic target.
@en
P2093
P2860
P304
P31
P356
P433
P478
P577
P698