Regulatory role of dendritic cells in postinfarction healing and left ventricular remodeling. - Wikidata - awgldk - TriplyDB

Regulatory role of dendritic cells in postinfarction healing and left ventricular remodeling.

Regulatory role of dendritic cells in postinfarction healing and left ventricular remodeling.

http://www.wikidata.org/entity/Q50503050

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Inflammation and Inflammatory Cells in Myocardial Infarction and Reperfusion Injury: A Double-Edged Sword Dendritic Cells and Their Role in Cardiovascular Diseases: A View on Human Studies Crosstalk between the heart and peripheral organs in heart failure Novel Molecular Approaches in Heart Failure: Seven Trans-Membrane Receptors Signaling in the Heart and Circulating Blood Leukocytes Communication in the heart: the role of the innate immune system in coordinating cellular responses to ischemic injury Phagocyte-myocyte interactions and consequences during hypoxic wound healing.Immune Modulation of Cardiac Repair and Regeneration: The Art of Mending Broken Hearts Opposite effects of gene deficiency and pharmacological inhibition of soluble epoxide hydrolase on cardiac fibrosis Role of T-cells in myocardial infarction.It takes two to tango: monocyte and macrophage duality in the infarcted heart Targeted injection of a biocomposite material alters macrophage and fibroblast phenotype and function following myocardial infarction: relation to left ventricular remodeling Leukocyte behavior in atherosclerosis, myocardial infarction, and heart failure.Synergistic effects of SDF-1α chemokine and hyaluronic acid release from degradable hydrogels on directing bone marrow derived cell homing to the myocardium.Lipopolysaccharide-induced M2 to M1 macrophage transformation for IL-12p70 production is blocked by Candida albicans mediated up-regulation of EBI3 expression.β-arrestin2 in infiltrated macrophages inhibits excessive inflammation after myocardial infarction Decreased myocardial dendritic cells is associated with impaired reparative fibrosis and development of cardiac rupture after myocardial infarction in humans.Chlamydia pneumoniae infection in mice induces chronic lung inflammation, iBALT formation, and fibrosis.The heme oxygenase system rescues hepatic deterioration in the condition of obesity co-morbid with type-2 diabetes Fibroblast growth factor-9 enhances M2 macrophage differentiation and attenuates adverse cardiac remodeling in the infarcted diabetic heart Relationship between neutrophil-to-lymphocyte ratio and electrocardiographic ischemia grade in STEMI.Myocardial infarction models in NOD/Scid mice for cell therapy research: permanent ischemia vs ischemia-reperfusion.Macrophage polarisation changes within the time between diagnostic biopsy and tumour resection in oral squamous cell carcinomas--an immunohistochemical study.Caveolin-1 deletion exacerbates cardiac interstitial fibrosis by promoting M2 macrophage activation in mice after myocardial infarction.Visualization of in vivo metabolic flows reveals accelerated utilization of glucose and lactate in penumbra of ischemic heart.Mononuclear Phagocytes Are Dispensable for Cardiac Remodeling in Established Pressure-Overload Heart Failure.CD11c(hi) Dendritic Cells Regulate Ly-6C(hi) Monocyte Differentiation to Preserve Immune-privileged CNS in Lethal Neuroinflammation.Myeloid mineralocorticoid receptor during experimental ischemic stroke: effects of model and sex.Deleterious effect of the IL-23/IL-17A axis and γδT cells on left ventricular remodeling after myocardial infarction.The impact of pressure overload on coronary vascular changes following myocardial infarction in rats Prognostic value of polarized macrophages in patients with hepatocellular carcinoma after curative resection.Monocyte and macrophage heterogeneity in the heart.The role of antioxidation and immunomodulation in postnatal multipotent stem cell-mediated cardiac repair.CD13 is essential for inflammatory trafficking and infarct healing following permanent coronary artery occlusion in mice.Enhanced efferocytosis of apoptotic cardiomyocytes through myeloid-epithelial-reproductive tyrosine kinase links acute inflammation resolution to cardiac repair after infarction.The risk of heart failure and cardiometabolic complications in obesity may be masked by an apparent healthy status of normal blood glucose.The heme oxygenase system suppresses perirenal visceral adiposity, abates renal inflammation and ameliorates diabetic nephropathy in Zucker diabetic fatty rats.Myocardial Immunocompetent Cells and Macrophage Phenotypes as Histopathological Surrogates for Diagnosis of Cardiac Sarcoidosis in Japanese.Reoxygenation-derived toxic reactive oxygen/nitrogen species modulate the contribution of bone marrow progenitor cells to remodeling after myocardial infarction.Hydrogen sulfide attenuates the recruitment of CD11b⁺Gr-1⁺ myeloid cells and regulates Bax/Bcl-2 signaling in myocardial ischemia injury.Myocardial Infarction Primes Autoreactive T Cells through Activation of Dendritic Cells.

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Regulatory role of dendritic cells in postinfarction healing and left ventricular remodeling.

http://www.wikidata.org/entity/Q50503050

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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name

Regulatory role of dendritic c ...... d left ventricular remodeling.

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Regulatory role of dendritic c ...... d left ventricular remodeling.

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type

label

Regulatory role of dendritic c ...... d left ventricular remodeling.

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Regulatory role of dendritic c ...... d left ventricular remodeling.

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prefLabel

Regulatory role of dendritic c ...... d left ventricular remodeling.

@en

Regulatory role of dendritic c ...... d left ventricular remodeling.

@nl

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CIRCULATIONAHA.111.052126

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Regulatory role of dendritic c ...... d left ventricular remodeling.

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Atsushi Anzai

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Hidehiro Kaneko

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Hitoshi Abe

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Keiichi Fukuda

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Kotaro Naito

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Motoaki Sano

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Satoshi Ogawa

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Satsuki Mochizuki

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Shigenori Nagai

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Toshihisa Anzai

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1234-1245

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scholarly article

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10.1161/CIRCULATIONAHA.111.052126

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2012-02-03T00:00:00Z

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