Valproate reduces CHOP levels and preserves oligodendrocytes and axons after spinal cord injury.
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Therapeutic potential of mood stabilizers lithium and valproic acid: beyond bipolar disorderNeuronal endoplasmic reticulum stress in axon injury and neurodegenerationFunctional Role of the Disulfide Isomerase ERp57 in Axonal RegenerationValproic Acid Induces Endocytosis-Mediated Doxorubicin Internalization and Shows Synergistic Cytotoxic Effects in Hepatocellular Carcinoma CellsValproate pretreatment protects pancreatic β-cells from palmitate-induced ER stress and apoptosis by inhibiting glycogen synthase kinase-3βValproic acid improves locomotion in vivo after SCI and axonal growth of neurons in vitroValproate Attenuates 25-kDa C-Terminal Fragment of TDP-43-Induced Neuronal Toxicity via Suppressing Endoplasmic Reticulum Stress and Activating Autophagy.The neuroprotective effect of treatment of valproic Acid in acute spinal cord injury.Rescue of Glaucomatous Neurodegeneration by Differentially Modulating Neuronal Endoplasmic Reticulum Stress Molecules.Efficacy of Anti-Epileptic Drugs in the Treatment of Tumor and Its Associated Epilepsy: An in vitro PerspectiveBone marrow stromal cells inhibit caspase-12 expression in rats with spinal cord injuryEndoplasmic reticulum stress: its role in disease and novel prospects for therapy.Valproic acid protects motor neuron death by inhibiting oxidative stress and endoplasmic reticulum stress-mediated cytochrome C release after spinal cord injuryThe effect of valproic acid on bovine oocyte maturation and early embryonic development in vitro.Valproate Attenuates Endoplasmic Reticulum Stress-Induced Apoptosis in SH-SY5Y Cells via the AKT/GSK3β Signaling Pathway.Epigenetic changes in patients with multiple sclerosis.Endoplasmic reticulum stress: relevance and therapeutics in central nervous system diseases.Valproic acid-mediated neuroprotection and neurogenesis after spinal cord injury: from mechanism to clinical potential.From demyelination to remyelination: the road toward therapies for spinal cord injury.Organotypic Spinal Cord Culture: a Proper Platform for the Functional Screening.Chaperone Proteins in the Central Nervous System and Peripheral Nervous System after Nerve Injury.De-repression of myelin-regulating gene expression after status epilepticus in mice lacking the C/EBP homologous protein CHOPActivation of the unfolded protein response enhances motor recovery after spinal cord injury.Endoplasmic Reticulum Stress and Disrupted Neurogenesis in the Brain Are Associated with Cognitive Impairment and Depressive-Like Behavior after Spinal Cord Injury.Valproic acid attenuates microgliosis in injured spinal cord and purinergic P2X4 receptor expression in activated microglia.Valproic acid attenuates blood-spinal cord barrier disruption by inhibiting matrix metalloprotease-9 activity and improves functional recovery after spinal cord injury.Age-related cataracts: Role of unfolded protein response, Ca2+ mobilization, epigenetic DNA modifications, and loss of Nrf2/Keap1 dependent cytoprotection.Oligodendrocyte precursor cell transplantation promotes functional recovery following contusive spinal cord injury in rats and is associated with altered microRNA expression.Epidermal neural crest stem cell-derived glia enhance neurotrophic elements in an ex vivo model of spinal cord injury.Decreased GFAP expression and improved functional recovery in contused spinal cord of rats following valproic acid therapy.Bog bilberry anthocyanin extract improves motor functional recovery by multifaceted effects in spinal cord injury.Upregulation of PTP1B After Rat Spinal Cord Injury.Valproic acid preserves motoneurons following contusion in organotypic spinal cord slice culture.Class I histone deacetylase (HDAC) inhibitor CI-994 promotes functional recovery following spinal cord injury.Valproic acid attenuates traumatic spinal cord injury-induced inflammation via STAT1 and NF-κB pathway dependent of HDAC3.Valproic Acid Attenuates Traumatic Brain Injury-Induced Inflammation in Vivo: Involvement of Autophagy and the Nrf2/ARE Signaling Pathway.
P2860
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P2860
Valproate reduces CHOP levels and preserves oligodendrocytes and axons after spinal cord injury.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh-hant
name
Valproate reduces CHOP levels ...... xons after spinal cord injury.
@en
Valproate reduces CHOP levels ...... xons after spinal cord injury.
@nl
type
label
Valproate reduces CHOP levels ...... xons after spinal cord injury.
@en
Valproate reduces CHOP levels ...... xons after spinal cord injury.
@nl
prefLabel
Valproate reduces CHOP levels ...... xons after spinal cord injury.
@en
Valproate reduces CHOP levels ...... xons after spinal cord injury.
@nl
P2093
P50
P1433
P1476
Valproate reduces CHOP levels ...... axons after spinal cord injury
@en
P2093
E Asensio-Pinilla
M S Guzmán-Lenis
P356
10.1016/J.NEUROSCIENCE.2011.01.012
P407
P577
2011-01-14T00:00:00Z