Gene expression profiling of peripheral blood from patients with untreated new-onset systemic juvenile idiopathic arthritis reveals molecular heterogeneity that may predict macrophage activation syndrome.
about
Horror autoinflammaticus: the molecular pathophysiology of autoinflammatory disease (*)Making sense of the cytokine storm: a conceptual framework for understanding, diagnosing, and treating hemophagocytic syndromesSystematic review: macrophage activation syndrome in inflammatory bowel diseaseMacrophage activation syndrome: advances towards understanding pathogenesisBiomarkers in systemic juvenile idiopathic arthritis: a comparison with biomarkers in cryopyrin-associated periodic syndromesLife-threatening complications of adult-onset Still's disease.Synovial fluid proteins differentiate between the subtypes of juvenile idiopathic arthritis.Assessing the human immune system through blood transcriptomics.Distribution of circulating cells in systemic juvenile idiopathic arthritis across disease activity states.A genomic approach to human autoimmune diseases.Biologic predictors of extension of oligoarticular juvenile idiopathic arthritis as determined from synovial fluid cellular composition and gene expression.Cytomegalovirus infection in pediatric rheumatic diseases: a review.Role of IL-1 beta in the development of human T(H)17 cells: lesson from NLPR3 mutated patients.Cell subset prediction for blood genomic studiesMonocytes are resistant to apoptosis in systemic juvenile idiopathic arthritis.Development of positive antinuclear antibodies and rheumatoid factor in systemic juvenile idiopathic arthritis points toward an autoimmune phenotype later in the disease course.Immature cell populations and an erythropoiesis gene-expression signature in systemic juvenile idiopathic arthritis: implications for pathogenesis.Pathogenesis of adult-onset Still's disease: new insights from the juvenile counterpart.Gene Expression Profiles from Peripheral Blood Mononuclear Cells Are Sensitive to Short Processing DelaysErythroid-specific transcriptional changes in PBMCs from pulmonary hypertension patientsAn activating NLRC4 inflammasome mutation causes autoinflammation with recurrent macrophage activation syndromePathogenesis of systemic juvenile idiopathic arthritis: some answers, more questions.The use of haplotype-specific transcripts improves sample annotation consistency.An overview of genetics of paediatric rheumatic diseases.Antibodies to MHC class II molecules induce autoimmunity: critical role for macrophages in the immunopathogenesis of obliterative airway disease.Correlation analyses of clinical and molecular findings identify candidate biological pathways in systemic juvenile idiopathic arthritisBiologic similarities based on age at onset in oligoarticular and polyarticular subtypes of juvenile idiopathic arthritis.[Caspase-1 regulates autoinflammation in rheumatic diseases].A phase II, multicenter, open-label study evaluating dosing and preliminary safety and efficacy of canakinumab in systemic juvenile idiopathic arthritis with active systemic features.A multicentre, randomised, double-blind, placebo-controlled trial with the interleukin-1 receptor antagonist anakinra in patients with systemic-onset juvenile idiopathic arthritis (ANAJIS trial)Gene expression profiling of peripheral blood mononuclear cells from children with active hemophagocytic lymphohistiocytosis.Repeated TLR9 stimulation results in macrophage activation syndrome-like disease in mice.Brief report: alternative activation of laser-captured murine hemophagocytes.How I treat hemophagocytic lymphohistiocytosis.Alternative activation in systemic juvenile idiopathic arthritis monocytesTargeting the inflammasome in rheumatic diseases.Molecular mechanisms in genetically defined autoinflammatory diseases: disorders of amplified danger signaling.Gene-expression analysis of adult-onset Still's disease and systemic juvenile idiopathic arthritis is consistent with a continuum of a single disease entity.The limited role of interferon-γ in systemic juvenile idiopathic arthritis cannot be explained by cellular hyporesponsivenessMacrophage activation syndrome in children with systemic lupus erythematosus and children with juvenile idiopathic arthritis
P2860
Q24631660-09724FB1-45CD-43CD-AA5C-3CD7811D065DQ27011625-DF4B72B3-A7D0-4058-99E6-9936182154A1Q27025349-AF5EB9A5-5AC8-4E4A-9324-F33BC011AE86Q28284000-0D22D85B-A139-47BF-B47D-8C287411CD5CQ28647737-511A7EFD-7F7F-425C-82C7-05CAB13EB0A5Q33412732-04F33F44-4934-47D1-8D2E-F7CF01F8F166Q33539890-B3526774-207F-4E2F-A5D3-B04BAE107167Q33629522-0CFB3B8A-C0A3-49FC-985F-C24C84B558FAQ33636807-965CA60C-BCEC-43D4-A996-E237261503F8Q33762839-3ECEC47A-BC1E-49F4-A10B-B1536B3F5986Q33815056-92ADCA1E-4316-4BF9-8998-FF90CC13BE4EQ33912101-873CF352-0AB9-4A40-B63A-27EEE0A572D8Q33920812-207F85FE-FFB5-42D7-B0C0-C13E92478C24Q33942400-086D1021-412E-438E-98F3-5B7442FD068CQ33987294-ACA6177C-2FC6-436D-876D-DAA7908EF615Q34023067-F51C45BB-670B-45EF-8F21-CC3CCBE3F6AFQ34027661-AEFA9EA5-24D8-4BAE-AF9D-0C5CBC043571Q34042413-4E94FC55-39C2-4E73-B234-8401ACB651BCQ34199302-4C93675A-72D0-4119-B0C6-70F2C52B5670Q34251071-6D94295A-E269-4155-A045-305C01EB39B3Q34254955-9ABA8687-477A-4349-803B-845325C17208Q34269827-AC8D4FDF-A083-4389-A465-7BB021FEC6B7Q34285017-59EE3CCE-1A48-4D4A-8030-354AA4BEB50CQ34363289-EB13FF4C-C433-44AF-954E-4BA1ADA7F627Q34382434-26BCFE2F-8D29-4F42-A708-DBDB9DCB9733Q34455674-68F4C4CB-AA8B-498E-8D14-167654FD1083Q34476320-794B06B9-814B-44E9-9CA3-24ED896058CEQ34520488-38C2F7EB-1501-402E-B95D-321D7D90C87CQ34632661-A34757B0-E180-44DD-934F-3B4FEBC7F949Q34758768-E05CC463-3A32-42D9-AF98-2FAAE63CE6D2Q34924532-6B6EC4A4-DF70-4F5C-B19A-4577C4F5E225Q35015661-03614113-9124-4513-901C-256CCF610FA5Q35195153-FBF6B028-937B-435A-8597-94DB36975BBBQ35483540-0B491DC9-4056-4DAB-BEF5-888CC622EDC7Q35784755-882FD21F-BD49-49D4-8AA1-467B2A21D181Q35804717-0E4C349C-F444-40B9-8E88-CDA5B7985669Q36041225-994D76F3-E0A8-4B66-AB1B-B3C923191E3CQ36304070-5227ADB0-B9D0-4FB2-AEBA-20B82F31695EQ36351200-5B37CB72-E072-40B4-B118-D5590435D4B2Q36420991-1D0ECC87-B8D1-4447-8953-1BB92ECD9FD9
P2860
Gene expression profiling of peripheral blood from patients with untreated new-onset systemic juvenile idiopathic arthritis reveals molecular heterogeneity that may predict macrophage activation syndrome.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Gene expression profiling of p ...... acrophage activation syndrome.
@en
Gene expression profiling of p ...... acrophage activation syndrome.
@nl
type
label
Gene expression profiling of p ...... acrophage activation syndrome.
@en
Gene expression profiling of p ...... acrophage activation syndrome.
@nl
prefLabel
Gene expression profiling of p ...... acrophage activation syndrome.
@en
Gene expression profiling of p ...... acrophage activation syndrome.
@nl
P2093
P2860
P356
P1476
Gene expression profiling of p ...... acrophage activation syndrome.
@en
P2093
Alexei A Grom
Beth S Gottlieb
David D Sherry
David N Glass
Judyann Olson
Lorie Luyrink
Michael Barnes
Ndate Fall
Norman T Ilowite
Robert A Colbert
P2860
P304
P356
10.1002/ART.22981
P577
2007-11-01T00:00:00Z