ATP5J and ATP5H Proactive Expression Correlates with Cardiomyocyte Mitochondrial Dysfunction Induced by Fluoride.

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Effect of Excess Iodide Intake on Salivary Glands in a Swiss Albino Mice Model.Effects of Fluoride on SOD and CAT in Testis and Epididymis of Mice.

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ATP5J and ATP5H Proactive Expression Correlates with Cardiomyocyte Mitochondrial Dysfunction Induced by Fluoride.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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ATP5J and ATP5H Proactive Expr ...... sfunction Induced by Fluoride.

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ATP5J and ATP5H Proactive Expr ...... sfunction Induced by Fluoride.

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ATP5J and ATP5H Proactive Expr ...... sfunction Induced by Fluoride.

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ATP5J and ATP5H Proactive Expr ...... sfunction Induced by Fluoride.

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ATP5J and ATP5H Proactive Expr ...... sfunction Induced by Fluoride.

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ATP5J and ATP5H Proactive Expr ...... sfunction Induced by Fluoride.

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P1476

ATP5J and ATP5H Proactive Expr ...... sfunction Induced by Fluoride.

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Bian-Hua Zhou

http://www.w3.org/2001/XMLSchema#string

Hong-Wei Wang

http://www.w3.org/2001/XMLSchema#string

Jing Liu

http://www.w3.org/2001/XMLSchema#string

Pan-Pan Tan

http://www.w3.org/2001/XMLSchema#string

Wei-Shun Tian

http://www.w3.org/2001/XMLSchema#string

Wen-Peng Zhao

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P2860

From ATP to PTP and Back: A Dual Function for the Mitochondrial ATP Synthase

Evaluation of the cardiolipin biosynthetic pathway and its interactions in the diabetic heart

ATP5H/KCTD2 locus is associated with Alzheimer's disease risk.

The role of mitochondria in reactive oxygen species metabolism and signaling.

Models of cardiac excitation-contraction coupling in ventricular myocytes

Over-expression of the ATP5J gene correlates with cell migration and 5-fluorouracil sensitivity in colorectal cancer.

Protein kinase C-α interaction with F0F1-ATPase promotes F0F1-ATPase activity and reduces energy deficits in injured renal cells.

The decreased expression of mitofusin-1 and increased fission-1 together with alterations in mitochondrial morphology in the kidney of rats with chronic fluorosis may involve elevated oxidative stress.

Structure of the cytosolic part of the subunit b-dimer of Escherichia coli F0F1-ATP synthase.

Expert consensus document: Mitochondrial function as a therapeutic target in heart failure

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ATP5J and ATP5H Proactive Expression Correlates with Cardiomyocyte Mitochondrial Dysfunction Induced by Fluoride.

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ATP5J and ATP5H Proactive Expression Correlates with Cardiomyocyte Mitochondrial Dysfunction Induced by Fluoride.

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