Relationship between exercise volume and muscle protein synthesis in a rat model of resistance exercise.

about

Relationship between exercise volume and muscle protein synthesis in a rat model of resistance exercise.

Item

http://www.wikidata.org/entity/Q50879152

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Relationship between exercise ...... model of resistance exercise.

@en

Relationship between exercise ...... model of resistance exercise.

@nl

type

Item

label

Relationship between exercise ...... model of resistance exercise.

@en

Relationship between exercise ...... model of resistance exercise.

@nl

prefLabel

Relationship between exercise ...... model of resistance exercise.

@en

Relationship between exercise ...... model of resistance exercise.

@nl

P1476

Relationship between exercise ...... model of resistance exercise.

@en

P2093

Junya Takegaki

http://www.w3.org/2001/XMLSchema#string

Koichi Nakazato

http://www.w3.org/2001/XMLSchema#string

Naokata Ishii

http://www.w3.org/2001/XMLSchema#string

Yuki Arihara

http://www.w3.org/2001/XMLSchema#string

P2860

Low-load high volume resistance exercise stimulates muscle protein synthesis more than high-load low volume resistance exercise in young men.

Novel insights into the regulation of skeletal muscle protein synthesis as revealed by a new nonradioactive in vivo technique

AMPK: a key sensor of fuel and energy status in skeletal muscle.

Aging Reduces the Activation of the mTORC1 Pathway after Resistance Exercise and Protein Intake in Human Skeletal Muscle: Potential Role of REDD1 and Impaired Anabolic Sensitivity

Emerging role for regulated in development and DNA damage 1 (REDD1) in the regulation of skeletal muscle metabolism.

The role of mTOR signalling in the regulation of skeletal muscle mass in a rodent model of resistance exercise.

Direct and macrophage-mediated actions of fatty acids causing insulin resistance in muscle cells.

Acute resistance exercise activates rapamycin-sensitive and -insensitive mechanisms that control translational activity and capacity in skeletal muscle.

Leucine-enriched essential amino acids attenuate muscle soreness and improve muscle protein synthesis after eccentric contractions in rats.

Rapamycin does not prevent increases in myofibrillar or mitochondrial protein synthesis following endurance exercise.

P304

710-716

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1152/JAPPLPHYSIOL.01009.2016

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

123

http://www.w3.org/2001/XMLSchema#string

P50

Riki Ogasawara

P577

2017-07-20T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28729395

http://www.w3.org/2001/XMLSchema#string