Overexpression of ALK5 Induces Human Tenon's Capsule Fibroblasts Transdifferentiation and Fibrosis In Vitro.

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Overexpression of ALK5 Induces Human Tenon's Capsule Fibroblasts Transdifferentiation and Fibrosis In Vitro.

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2017年の論文

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Overexpression of ALK5 Induces ...... tiation and Fibrosis In Vitro.

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Overexpression of ALK5 Induces ...... tiation and Fibrosis In Vitro.

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Overexpression of ALK5 Induces ...... tiation and Fibrosis In Vitro.

@en

Overexpression of ALK5 Induces ...... tiation and Fibrosis In Vitro.

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Overexpression of ALK5 Induces ...... tiation and Fibrosis In Vitro.

@en

Overexpression of ALK5 Induces ...... tiation and Fibrosis In Vitro.

@nl

P1476

Overexpression of ALK5 Induces ...... tiation and Fibrosis In Vitro.

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P2093

Hui-Min Shi

http://www.w3.org/2001/XMLSchema#string

Hui-Ying Wang

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Jia-Ying Zhang

http://www.w3.org/2001/XMLSchema#string

Nan Zhang

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Shu-Hao Fu

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Wen Ye

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Yi-Qin Xiao

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P2860

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L- and S-endoglin differentially modulate TGFbeta1 signaling mediated by ALK1 and ALK5 in L6E9 myoblasts

Urotensin II induces phenotypic differentiation, migration, and collagen synthesis of adventitial fibroblasts from rat aorta

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Transforming growth factor-beta signaling through the Smad pathway: role in extracellular matrix gene expression and regulation.

A portrait of Transforming Growth Factor β superfamily signalling: Background matters.

Enhanced trabeculectomy: the Moorfields Safer Surgery System.

Wound modulation after filtration surgery.

IN-1130, a novel transforming growth factor-beta type I receptor kinase (ALK5) inhibitor, suppresses renal fibrosis in obstructive nephropathy.

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1018-1028

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scholarly article

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10.1080/02713683.2016.1276198

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P478

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2017-02-28T00:00:00Z

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28632033

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P921

overexpression