Anti-tumour necrosis factor therapy enhances mucosal healing through down-regulation of interleukin-21 expression and T helper type 17 cell infiltration in Crohn's disease.
about
Molecular mechanism of action of anti-tumor necrosis factor antibodies in inflammatory bowel diseasesEctopic Tertiary Lymphoid Tissue in Inflammatory Bowel Disease: Protective or Provocateur?Infliximab preferentially induces clinical remission and mucosal healing in short course Crohn's disease with luminal lesions through balancing abnormal immune response in gut mucosaSerum Levels of Lipopolysaccharide and 1,3-β-D-Glucan Refer to the Severity in Patients with Crohn's Disease.Inflammatory Bowel Disease: How Effective Is TNF-α Suppression?Genetic Polymorphisms of IL-17F and TRAF3IP2 Could Be Predictive Factors of the Long-Term Effect of Infliximab against Crohn's DiseaseDivalent metal-ion transporter 1 is decreased in intestinal epithelial cells and contributes to the anemia in inflammatory bowel disease.Blockade of PLD2 Ameliorates Intestinal Mucosal Inflammation of Inflammatory Bowel DiseaseImmunopathogenesis of granulomas in chronic autoinflammatory diseases.Dysregulation of mucosal immune response in pathogenesis of inflammatory bowel disease.Stopping anti-TNF agents in patients with Crohn's disease in remission: is it a feasible long-term strategy?New Insights into the Mechanisms of Action of Anti-Tumor Necrosis Factor-α Monoclonal Antibodies in Inflammatory Bowel Disease.Hypoxia inducible factor-1α-induced interleukin-33 expression in intestinal epithelia contributes to mucosal homeostasis in inflammatory bowel disease.Genetic Markers Predict Primary Non-Response and Durable Response To Anti-TNF Biologic Therapies in Crohn's Disease.Global mucosal and serum cytokine profile in patients with ulcerative colitis undergoing anti-TNF therapy.Serum bacterial toxins are related to the progression of inflammatory bowel disease.CD99 refers to the activity of inflammatory bowel disease.Effects of Anti-TNFα Treatment on Mucosal Expression of IL-17A, IL-21, and IL-22 and Cytokine-Producing T Cell Subsets in Crohn's Disease.Evaluating IL-21 as a Potential Therapeutic Target in Crohn's Disease.Circulating CD4(+)CXCR5(+) T Cells Exacerbate B Cell Antibody Production in Crohn's Disease Through IL-21 SecretionIdentification of inflammatory mediators in patients with Crohn's disease unresponsive to anti-TNFα therapy
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P2860
Anti-tumour necrosis factor therapy enhances mucosal healing through down-regulation of interleukin-21 expression and T helper type 17 cell infiltration in Crohn's disease.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
@wuu
2013年学术文章
@zh
2013年学术文章
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2013年学术文章
@zh-hans
2013年学术文章
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2013年学术文章
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name
Anti-tumour necrosis factor th ...... filtration in Crohn's disease.
@en
Anti-tumour necrosis factor th ...... filtration in Crohn's disease.
@nl
type
label
Anti-tumour necrosis factor th ...... filtration in Crohn's disease.
@en
Anti-tumour necrosis factor th ...... filtration in Crohn's disease.
@nl
prefLabel
Anti-tumour necrosis factor th ...... filtration in Crohn's disease.
@en
Anti-tumour necrosis factor th ...... filtration in Crohn's disease.
@nl
P2093
P2860
P356
P1476
Anti-tumour necrosis factor th ...... nfiltration in Crohn's disease
@en
P2093
P2860
P304
P356
10.1111/CEI.12084
P50
P577
2013-07-01T00:00:00Z