about
Mimicry by asx- and ST-turns of the four main types of beta-turn in proteins.Recurring main-chain anion-binding motifs in short polypeptides: nests.Atmospheric oxygen tension slows myoblast proliferation via mitochondrial activationPotential of oligonucleotide-mediated exon-skipping therapy for Duchenne muscular dystrophy.The isolated muscle fibre as a model of disuse atrophy: characterization using PhAct, a method to quantify f-actin.In silico screening based on predictive algorithms as a design tool for exon skipping oligonucleotides in Duchenne muscular dystrophyCellWhere: graphical display of interaction networks organized on subcellular localizations.Muscular dystrophy in the mdx mouse is a severe myopathy compounded by hypotrophy, hypertrophy and hyperplasia.Skeletal muscle characteristics are preserved in hTERT/cdk4 human myogenic cell lines.Changes in Communication between Muscle Stem Cells and their Environment with Aging.Exon skipping for nonsense mutations in Duchenne muscular dystrophy: too many mutations, too few patients?Dystrophin deficiency leads to disturbance of LAMP1-vesicle-associated protein secretion.Age-Associated Methylation Suppresses SPRY1, Leading to a Failure of Re-quiescence and Loss of the Reserve Stem Cell Pool in Elderly Muscle.Quantitative Antisense Screening and Optimization for Exon 51 Skipping in Duchenne Muscular Dystrophy.Annexin A2 links poor myofiber repair with inflammation and adipogenic replacement of the injured muscle.Antisense PMO cocktails effectively skip dystrophin exons 45-55 in myotubes transdifferentiated from DMD patient fibroblasts.Identification of Novel Antisense-Mediated Exon Skipping Targets in DYSF for Therapeutic Treatment of DysferlinopathyPersonalized Medicine and Molecular Interaction Networks in Amyotrophic Lateral Sclerosis (ALS): Current KnowledgeMuscle Gene Sets: a versatile methodological aid to functional genomics in the neuromuscular fieldA Systematic Review of Suggested Molecular Strata, Biomarkers and Their Tissue Sources in ALSDirect Reprogramming of Human DMD Fibroblasts into Myotubes for In Vitro Evaluation of Antisense-Mediated Exon Skipping and Exons 45-55 Skipping Accompanied by Rescue of Dystrophin Expression.Exons 45-55 Skipping Using Mutation-Tailored Cocktails of Antisense Morpholinos in the DMD GeneMyoMiner: explore gene co-expression in normal and pathological muscle
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description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
William Duddy
@ast
William Duddy
@en
William Duddy
@es
William Duddy
@nl
William Duddy
@sl
type
label
William Duddy
@ast
William Duddy
@en
William Duddy
@es
William Duddy
@nl
William Duddy
@sl
prefLabel
William Duddy
@ast
William Duddy
@en
William Duddy
@es
William Duddy
@nl
William Duddy
@sl
P106
P21
P31
P496
0000-0003-2239-9094