Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophy.
about
Integration of flux measurements to resolve changes in anabolic and catabolic metabolism in cardiac myocytesA proteolytic fragment of histone deacetylase 4 protects the heart from failure by regulating the hexosamine biosynthetic pathway.AMPK activation counteracts cardiac hypertrophy by reducing O-GlcNAcylation.Gene expression analysis to identify mechanisms underlying heart failure susceptibility in mice and humans.Cardiac-specific overexpression of aldehyde dehydrogenase 2 exacerbates cardiac remodeling in response to pressure overload.
P2860
Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophy.
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Cardiomyocyte Ogt limits ventr ...... without affecting hypertrophy.
@en
Cardiomyocyte Ogt limits ventr ...... without affecting hypertrophy.
@nl
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label
Cardiomyocyte Ogt limits ventr ...... without affecting hypertrophy.
@en
Cardiomyocyte Ogt limits ventr ...... without affecting hypertrophy.
@nl
prefLabel
Cardiomyocyte Ogt limits ventr ...... without affecting hypertrophy.
@en
Cardiomyocyte Ogt limits ventr ...... without affecting hypertrophy.
@nl
P2093
P2860
P1476
Cardiomyocyte Ogt limits ventr ...... without affecting hypertrophy.
@en
P2093
Allison L Aird
Angelica M DeMartino
Anna M Gumpert
Bethany W Long
Kenneth R Brittian
Lewis J Watson
Peter J Kilfoil
Robert E Brainard
Senthilkumar Muthusamy
Sujith Dassanayaka
P2860
P2888
P356
10.1007/S00395-017-0612-7
P577
2017-03-15T00:00:00Z