Synaptotagmin-2, and -1, linked to neurotransmission impairment and vulnerability in Spinal Muscular Atrophy.
about
Bioenergetic status modulates motor neuron vulnerability and pathogenesis in a zebrafish model of spinal muscular atrophy.Smn-Deficiency Increases the Intrinsic Excitability of Motoneurons.BDNF/trkB Induction of Calcium Transients through Cav2.2 Calcium Channels in Motoneurons Corresponds to F-actin Assembly and Growth Cone Formation on β2-Chain Laminin (221).Transmitter release site organization can predict synaptic function at the neuromuscular junction.Engineering Botulinum Toxins to Improve and Expand Targeting and SNARE Cleavage Activity
P2860
Synaptotagmin-2, and -1, linked to neurotransmission impairment and vulnerability in Spinal Muscular Atrophy.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
@zh
2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
@yue
2016年學術文章
@zh-hant
name
Synaptotagmin-2, and -1, linke ...... ty in Spinal Muscular Atrophy.
@en
Synaptotagmin-2, and -1, linke ...... ty in Spinal Muscular Atrophy.
@nl
type
label
Synaptotagmin-2, and -1, linke ...... ty in Spinal Muscular Atrophy.
@en
Synaptotagmin-2, and -1, linke ...... ty in Spinal Muscular Atrophy.
@nl
prefLabel
Synaptotagmin-2, and -1, linke ...... ty in Spinal Muscular Atrophy.
@en
Synaptotagmin-2, and -1, linke ...... ty in Spinal Muscular Atrophy.
@nl
P2860
P356
P1476
Synaptotagmin-2, and -1, linke ...... ity in Spinal Muscular Atrophy
@en
P2093
Lucía Tabares
Saravanan Arumugam
P2860
P304
P356
10.1093/HMG/DDW297
P577
2016-11-01T00:00:00Z