DNase Sda1 provides selection pressure for a switch to invasive group A streptococcal infection. - Wikidata - awgldk - TriplyDB

DNase Sda1 provides selection pressure for a switch to invasive group A streptococcal infection.

DNase Sda1 provides selection pressure for a switch to invasive group A streptococcal infection.

http://www.wikidata.org/entity/Q51185403

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Gene repertoire evolution of Streptococcus pyogenes inferred from phylogenomic analysis with Streptococcus canis and Streptococcus dysgalactiae Microevolution of group A streptococci in vivo: capturing regulatory networks engaged in sociomicrobiology, niche adaptation, and hypervirulence Functional analysis of the group A streptococcal luxS/AI-2 system in metabolism, adaptation to stress and interaction with host cells TrxR, a new CovR-repressed response regulator that activates the Mga virulence regulon in group A Streptococcus Do neutrophil extracellular traps contribute to the heightened risk of thrombosis in inflammatory diseases?Molecular epidemiology and genomics of group A Streptococcus PAMPs and DAMPs as triggers for DIC The structural characterization of a prophage-encoded extracellular DNase from Streptococcus pyogenes Ultrahigh and High Resolution Structures and Mutational Analysis of Monomeric Streptococcus pyogenes SpeB Reveal a Functional Role for the Glycine-rich C-terminal Loop Neutrophil Extracellular Traps Go Viral Neutrophils: Between host defence, immune modulation, and tissue injury Molecular and genomic characterization of pathogenic traits of group A Streptococcus pyogenes Dispersal of Group A streptococcal biofilms by the cysteine protease SpeB leads to increased disease severity in a murine model A Two-Component Regulatory System Impacts Extracellular Membrane-Derived Vesicle Production in Group A Streptococcus Regulation of CovR expression in Group B Streptococcus impacts blood-brain barrier penetration.The TatD-like DNase of Plasmodium is a virulence factor and a potential malaria vaccine candidate Evolutionary Constraints Shaping Streptococcus pyogenes-Host Interactions.Tracing the evolutionary history of the pandemic group A streptococcal M1T1 clone.Complete Genome Sequence of emm1 Streptococcus pyogenes A20, a Strain with an Intact Two-Component System, CovRS, Isolated from a Patient with Necrotizing Fasciitis.ETosis: A Microbicidal Mechanism beyond Cell Death.Neutrophil extracellular traps: is immunity the second function of chromatin?Infection-induced NETosis is a dynamic process involving neutrophil multitasking in vivo.An unbiased systems genetics approach to mapping genetic loci modulating susceptibility to severe streptococcal sepsis.Incompetence of neutrophils to invasive group A streptococcus is attributed to induction of plural virulence factors by dysfunction of a regulator.A naturally occurring mutation in ropB suppresses SpeB expression and reduces M1T1 group A streptococcal systemic virulence.Inactivation of DltA modulates virulence factor expression in Streptococcus pyogenes A combination of independent transcriptional regulators shapes bacterial virulence gene expression during infection Highly frequent mutations in negative regulators of multiple virulence genes in group A streptococcal toxic shock syndrome isolates.DrsG from Streptococcus dysgalactiae subsp. equisimilis inhibits the antimicrobial peptide LL-37.A genomic perspective on a new bacterial genus and species from the Alcaligenaceae family, Basilea psittacipulmonis Emerging role of the interleukin-8 cleaving enzyme SpyCEP in clinical Streptococcus pyogenes infection Wiring the streptococcal network for alternative lifestyles Differences in SpeB protease activity among group A streptococci associated with superficial, invasive, and autoimmune disease.Streptococcal collagen-like protein A and general stress protein 24 are immunomodulating virulence factors of group A Streptococcus.Allelic replacement of the streptococcal cysteine protease SpeB in a Δsrv mutant background restores biofilm formation.M1T1 group A streptococcal pili promote epithelial colonization but diminish systemic virulence through neutrophil extracellular entrapment.Streptolysin S contributes to group A streptococcal translocation across an epithelial barrier.Genetic switch to hypervirulence reduces colonization phenotypes of the globally disseminated group A streptococcus M1T1 clone.Study of the IgG endoglycosidase EndoS in group A streptococcal phagocyte resistance and virulence.Chemokine-cleaving Streptococcus pyogenes protease SpyCEP is necessary and sufficient for bacterial dissemination within soft tissues and the respiratory tract.

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P2860

DNase Sda1 provides selection pressure for a switch to invasive group A streptococcal infection.

http://www.wikidata.org/entity/Q51185403

description

2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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DNase Sda1 provides selection ...... oup A streptococcal infection.

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DNase Sda1 provides selection ...... oup A streptococcal infection.

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DNase Sda1 provides selection ...... oup A streptococcal infection.

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DNase Sda1 provides selection ...... oup A streptococcal infection.

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DNase Sda1 provides selection ...... oup A streptococcal infection.

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DNase Sda1 provides selection ...... oup A streptococcal infection.

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DNase Sda1 provides selection ...... oup A streptococcal infection.

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Amelia J Simpson

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Andrew Hollands

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Gursharan S Chhatwal

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Jason D McArthur

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Jason N Cole

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John T Buchanan

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Joshua K Kirk

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Katrin Dinkla

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Malak Kotb

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10.1038/NM1612

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Anna Henningham

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2007-07-15T00:00:00Z

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