Maternal obesity eliminates the neonatal lamb plasma leptin peak.
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Early Life Nutrition and Energy Balance Disorders in Offspring in Later LifeEndocrine and other physiologic modulators of perinatal cardiomyocyte endowmentLeptin in pregnancy and development: a contributor to adulthood disease?Interventions to prevent adverse fetal programming due to maternal obesity during pregnancy.The impact of maternal overnutrition and obesity on hypothalamic-pituitary-adrenal axis response of offspring to stressMaternal obesity and developmental programming of metabolic disorders in offspring: evidence from animal models.Multigenerational impact of maternal overnutrition/obesity in the sheep on the neonatal leptin surge in granddaughters.Maternal obesity in sheep increases fatty acid synthesis, upregulates nutrient transporters, and increases adiposity in adult male offspring after a feeding challenge.Developmental programming in response to maternal overnutritionMaternal Obesity and the Fetal Origins of the Metabolic Syndrome.Elevated glucocorticoids during ovine pregnancy increase appetite and produce glucose dysregulation and adiposity in their granddaughters in response to ad libitum feeding at 1 year of age.Leptin in early life: a key factor for the development of the adult metabolic profile.Nutritional manipulations in the perinatal period program adipose tissue in offspring.Mechanisms by which maternal obesity programs offspring for obesity: evidence from animal studies.Maternal obesity upregulates fatty acid and glucose transporters and increases expression of enzymes mediating fatty acid biosynthesis in fetal adipose tissue depots.Maternal obesity programs reduced leptin signaling in the pituitary and altered GH/IGF1 axis function leading to increased adiposity in adult sheep offspring.Experimental Models of Maternal Obesity and Neuroendocrine Programming of Metabolic Disorders in OffspringPHYSIOLOGY AND ENDOCRINOLOGY SYMPOSIUM:The effects of poor maternal nutrition during gestation on offspring postnatal growth and metabolism.The effects of poor maternal nutrition during gestation on postnatal growth and development of lambs.The human obesity epidemic, the mismatch paradigm, and our modern "captive" environment.Uterine artery leptin receptors during the ovarian cycle and pregnancy regulate angiogenesis in ovine uterine artery endothelial cells†.
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P2860
Maternal obesity eliminates the neonatal lamb plasma leptin peak.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh-hant
name
Maternal obesity eliminates the neonatal lamb plasma leptin peak.
@en
Maternal obesity eliminates the neonatal lamb plasma leptin peak.
@nl
type
label
Maternal obesity eliminates the neonatal lamb plasma leptin peak.
@en
Maternal obesity eliminates the neonatal lamb plasma leptin peak.
@nl
prefLabel
Maternal obesity eliminates the neonatal lamb plasma leptin peak.
@en
Maternal obesity eliminates the neonatal lamb plasma leptin peak.
@nl
P2860
P1476
Maternal obesity eliminates the neonatal lamb plasma leptin peak.
@en
P2093
Nathan M Long
Stephen P Ford
P2860
P304
P356
10.1113/JPHYSIOL.2010.201681
P407
P577
2011-01-24T00:00:00Z