TLR4-mediated inflammation is a key pathogenic event leading to kidney damage and fibrosis in cyclosporine nephrotoxicity.
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Toll-like Receptor 4 Deficiency Reduces Oxidative Stress and Macrophage Mediated Inflammation in Hypertensive Kidney.Downregulation of kidney protective factors by inflammation: role of transcription factors and epigenetic mechanisms.Kidney Androgen-Regulated Protein (KAP) Transgenic Mice Are Protected Against High-Fat Diet Induced Metabolic Syndrome.Zoledronate dysregulates fatty acid metabolism in renal tubular epithelial cells to induce nephrotoxicity.U-box ubiquitin ligase PPIL2 suppresses breast cancer invasion and metastasis by altering cell morphology and promoting SNAI1 ubiquitination and degradation.
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TLR4-mediated inflammation is a key pathogenic event leading to kidney damage and fibrosis in cyclosporine nephrotoxicity.
description
2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年學術文章
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2016年學術文章
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name
TLR4-mediated inflammation is ...... n cyclosporine nephrotoxicity.
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TLR4-mediated inflammation is ...... n cyclosporine nephrotoxicity.
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type
label
TLR4-mediated inflammation is ...... n cyclosporine nephrotoxicity.
@en
TLR4-mediated inflammation is ...... n cyclosporine nephrotoxicity.
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TLR4-mediated inflammation is ...... n cyclosporine nephrotoxicity.
@en
TLR4-mediated inflammation is ...... n cyclosporine nephrotoxicity.
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P2093
P2860
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TLR4-mediated inflammation is ...... n cyclosporine nephrotoxicity.
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P2093
Adrián M Ramos
Consuelo Guerri
Cristian González-Guerrero
Jesús Egido
Pablo Cannata-Ortiz
P2860
P2888
P304
P356
10.1007/S00204-016-1830-8
P577
2016-09-01T00:00:00Z