Oxidative stress-mediated up-regulation of myocardial ischemic preconditioning up-regulated protein 1 gene expression in H9c2 cardiomyocytes is regulated by cyclic AMP-response element binding protein.

Oxidative stress-mediated up-regulation of myocardial ischemic preconditioning up-regulated protein 1 gene expression in H9c2 cardiomyocytes is regulated by cyclic AMP-response element binding protein.

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http://www.wikidata.org/entity/Q51557968
Constitutive heat shock protein 70 interacts with α-enolase and protects cardiomyocytes against oxidative stressMipu1 protects H9c2 myogenic cells from hydrogen peroxide-induced apoptosis through inhibition of the expression of the death receptor Fas.ZNF667/Mipu1 is a novel anti-apoptotic factor that directly regulates the expression of the rat Bax gene in H9c2 cellsMipu1 overexpression protects macrophages from oxLDL-induced foam cell formation and cell apoptosisMyocardial ischemic preconditioning upregulated protein 1(Mipu1):zinc finger protein 667 - a multifunctional KRAB/C2H2 zinc finger protein.Response of rat lung tissue to short-term hyperoxia: a proteomic approach.Mipu1, a novel direct target gene, is involved in hypoxia inducible factor 1-mediated cytoprotection.Identification of cardioprotective agents from traditional Chinese medicine against oxidative damage.Post-transcriptional regulation of placenta growth factor mRNA by hydrogen peroxide.Phenylephrine preconditioning in embryonic heart H9c2 cells is mediated by up-regulation of SUR2B/Kir6.2: A first evidence for functional role of SUR2B in sarcolemmal KATP channels and cardioprotection.MicroRNA-135a Regulates Apoptosis Induced by Hydrogen Peroxide in Rat Cardiomyoblast Cells.WDR26 promotes mitophagy of cardiomyocytes induced by hypoxia through Parkin translocation.The Protective Role of the TOPK/PBK Pathway in Myocardial Ischemia/Reperfusion and H₂O₂-Induced Injury in H9C2 Cardiomyocytes.miR-19a protects cardiomyocytes from hypoxia/reoxygenation-induced apoptosis via PTEN/PI3K/p-Akt pathway.Tumor necrosis factor alpha protects heart cultures against hypoxic damage via activation of PKA and phospholamban to prevent calcium overload.Insulin down-regulates cardioprotective SUR2A in the heart-derived H9c2 cells: A possible explanation for some adverse effects of insulin therapy
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Oxidative stress-mediated up-regulation of myocardial ischemic preconditioning up-regulated protein 1 gene expression in H9c2 cardiomyocytes is regulated by cyclic AMP-response element binding protein.

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http://www.wikidata.org/entity/Q51557968
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
Oxidative stress-mediated up-r ...... ponse element binding protein.
@en
Oxidative stress-mediated up-r ...... ponse element binding protein.
@nl
type
label
Oxidative stress-mediated up-r ...... ponse element binding protein.
@en
Oxidative stress-mediated up-r ...... ponse element binding protein.
@nl
prefLabel
Oxidative stress-mediated up-r ...... ponse element binding protein.
@en
Oxidative stress-mediated up-r ...... ponse element binding protein.
@nl
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Oxidative stress-mediated up-r ...... ponse element binding protein.
@en
P2093
P304
P31
P356
10.1016/J.FREERADBIOMED.2010.05.004
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P698