Overexpression of miR-210, a downstream target of HIF1α, causes centrosome amplification in renal carcinoma cells. - Wikidata - awgldk - TriplyDB

Overexpression of miR-210, a downstream target of HIF1α, causes centrosome amplification in renal carcinoma cells.

Overexpression of miR-210, a downstream target of HIF1α, causes centrosome amplification in renal carcinoma cells.

http://www.wikidata.org/entity/Q51582002

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New Concepts in Cancer Biomarkers: Circulating miRNAs in Liquid Biopsies Epigenomics of clear cell renal cell carcinoma: mechanisms and potential use in molecular pathology HypoxamiRs and cancer: from biology to targeted therapy OxymiRs in cutaneous development, wound repair and regeneration Noncoding RNA Profiles in Tobacco- and Alcohol-Associated Diseases Multiple functions of hypoxia-regulated miR-210 in cancer.High-throughput sequencing of microRNAs in peripheral blood mononuclear cells: identification of potential weight loss biomarkers.MiR-210 expression reverses radioresistance of stem-like cells of oesophageal squamous cell carcinoma.Epigenetic programming of hypoxic-ischemic encephalopathy in response to fetal hypoxia.Elevated expression of miR-210 predicts poor survival of cancer patients: a systematic review and meta-analysis.Genetic variants in microRNA machinery genes are associated [corrected] with idiopathic recurrent pregnancy loss risk.Increase of microRNA-210, decrease of raptor gene expression and alteration of mammalian target of rapamycin regulated proteins following mithramycin treatment of human erythroid cells.A clinical overview of centrosome amplification in human cancers.miR-210: fine-tuning the hypoxic response.Sexual dimorphism in miR-210 expression and mitochondrial dysfunction in the placenta with maternal obesity.MIR-210 modulates mitochondrial respiration in placenta with preeclampsia.miR-125b promotes cell death by targeting spindle assembly checkpoint gene MAD1 and modulating mitotic progression Elevation of Circulating miR-210-3p in High-Altitude Hypoxic Environment.MiR-210 disturbs mitotic progression through regulating a group of mitosis-related genes miR-210 is a target of hypoxia-inducible factors 1 and 2 in renal cancer, regulates ISCU and correlates with good prognosis.Reactive oxygen species-responsive miR-210 regulates proliferation and migration of adipose-derived stem cells via PTPN2 The regulation and function of microRNAs in kidney diseases.MicroRNA-210 induces apoptosis in colorectal cancer via induction of reactive oxygen.Analysis of MicroRNAs in sputum to improve computed tomography for lung cancer diagnosis.miR-210: the master hypoxamir.miR-210: a therapeutic target in cancer.Tumor hypoxia as a driving force in genetic instability.Emerging roles of miR-210 and other non-coding RNAs in the hypoxic response.Biomarkers in renal cancer.An emerging understanding of long noncoding RNAs in kidney cancer.MicroRNAs in renal cell carcinoma: a systematic review of clinical implications (Review).The role of hypoxia-induced miR-210 in cancer progression.Aberrantly expressed microRNAs in bladder cancer and renal cell carcinoma.MicroRNA-210 and its theranostic potential.WSS25 inhibits Dicer, downregulating microRNA-210, which targets Ephrin-A3, to suppress human microvascular endothelial cell (HMEC-1) tube formation.Prognostic value of meta-signature miRNAs in renal cell carcinoma: an integrated miRNA expression profiling analysis An integrated analysis of differential miRNA and mRNA expressions in human gallstones.Analysis of long non-coding RNA expression profiles in clear cell renal cell carcinoma.MiR-99b-5p expression and response to tyrosine kinase inhibitor treatment in clear cell renal cell carcinoma patients.MiR-210 is induced by Oct-2, regulates B cells, and inhibits autoantibody production.

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Overexpression of miR-210, a downstream target of HIF1α, causes centrosome amplification in renal carcinoma cells.

http://www.wikidata.org/entity/Q51582002

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Overexpression of miR-210, a d ...... tion in renal carcinoma cells.

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Overexpression of miR-210, a d ...... tion in renal carcinoma cells.

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Overexpression of miR-210, a d ...... tion in renal carcinoma cells.

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Overexpression of miR-210, a d ...... tion in renal carcinoma cells.

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Overexpression of miR-210, a d ...... tion in renal carcinoma cells.

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Overexpression of miR-210, a d ...... tion in renal carcinoma cells.

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The Journal of Pathology

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Overexpression of miR-210, a d ...... tion in renal carcinoma cells.

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Chisato Nakada

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Fuminori Sato

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Hiromitsu Mimata

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Keiko Matsuura

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Masao Seto

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Masatsugu Moriyama

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Naoki Hijiya

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Tomohisa Uchida

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Tung Lam Nguyen

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Yoshiyuki Tsukamoto

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P2860

MicroRNA-210 regulates mitochondrial free radical response to hypoxia and krebs cycle in cancer cells by targeting iron sulfur cluster protein ISCU

The tumour suppressor protein VHL targets hypoxia-inducible factors for oxygen-dependent proteolysis

Centrosome amplification drives chromosomal instability in breast tumor development.

MicroRNA-210 modulates endothelial cell response to hypoxia and inhibits the receptor tyrosine kinase ligand Ephrin-A3

MicroRNA-210 controls mitochondrial metabolism during hypoxia by repressing the iron-sulfur cluster assembly proteins ISCU1/2

A microRNA signature of hypoxia

VHL mutations and their correlation with tumour cell proliferation, microvessel density, and patient prognosis in clear cell renal cell carcinoma.

MicroRNA signatures in human cancers

MicroRNA regulation of DNA repair gene expression in hypoxic stress

Systematic sequencing of renal carcinoma reveals inactivation of histone modifying genes

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