TLR Tolerance as a Treatment for Central Nervous System Autoimmunity.

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Autoimmunity in 2016.The hygiene hypothesis in autoimmunity: the role of pathogens and commensals.Toll-like receptor 4 modulation influences human neural stem cell proliferation and differentiation.

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TLR Tolerance as a Treatment for Central Nervous System Autoimmunity.

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http://www.wikidata.org/entity/Q51583877

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

@zh-cn

2016年学术文章

@zh-hans

2016年学术文章

@zh-my

2016年学术文章

@zh-sg

2016年學術文章

@yue

2016年學術文章

@zh-hant

name

TLR Tolerance as a Treatment for Central Nervous System Autoimmunity.

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TLR Tolerance as a Treatment for Central Nervous System Autoimmunity.

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type

Item

label

TLR Tolerance as a Treatment for Central Nervous System Autoimmunity.

@en

TLR Tolerance as a Treatment for Central Nervous System Autoimmunity.

@nl

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TLR Tolerance as a Treatment for Central Nervous System Autoimmunity.

@en

TLR Tolerance as a Treatment for Central Nervous System Autoimmunity.

@nl

P1476

TLR Tolerance as a Treatment for Central Nervous System Autoimmunity

@en

P2093

Emily J Anstadt

http://www.w3.org/2001/XMLSchema#string

Frank Nichols

http://www.w3.org/2001/XMLSchema#string

Nicholas Wasko

http://www.w3.org/2001/XMLSchema#string

Robert B Clark

http://www.w3.org/2001/XMLSchema#string

P2860

Toll-like receptor 2 signaling in CD4(+) T lymphocytes promotes T helper 17 responses and regulates the pathogenesis of autoimmune disease.

Hyaluronan blocks oligodendrocyte progenitor maturation and remyelination through TLR2

TLR2-dependent activation of β-catenin pathway in dendritic cells induces regulatory responses and attenuates autoimmune inflammation.

Innate immunity mediated by TLR9 modulates pathogenicity in an animal model of multiple sclerosis.

Bacterial lipodipeptide, Lipid 654, is a microbiome-associated biomarker for multiple sclerosis

Signaling via the RIP2 adaptor protein in central nervous system-infiltrating dendritic cells promotes inflammation and autoimmunity.

Streptococcus pneumoniae Infection aggravates experimental autoimmune encephalomyelitis via Toll-like receptor 2.

Borrelia burgdorferi-induced tolerance as a model of persistence via immunosuppression.

Toll-like receptor 4 signaling in T cells promotes autoimmune inflammation

Myelin-specific regulatory T cells accumulate in the CNS but fail to control autoimmune inflammation.

P304

2110-2118

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scholarly article

P356

10.4049/JIMMUNOL.1600876

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

197

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P50

Mai Fujiwara

P577

2016-08-08T00:00:00Z

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27503211

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P921

Toll-like receptor

TLR Tolerance as a Treatment for Central Nervous System Autoimmunity.

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P2860

P2860

TLR Tolerance as a Treatment for Central Nervous System Autoimmunity.

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P1476

P2093

P2860

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P921