Protein Prenylation Constitutes an Endogenous Brake on Axonal Growth.
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Reconnecting Eye to Brain.Targeting 14-3-3 adaptor protein-protein interactions to stimulate central nervous system repair.a-Factor Analogues Containing Alkyne- and Azide-Functionalized Isoprenoids Are Efficiently Enzymatically Processed and Retain Wild-Type Bioactivity.Maximizing functional axon repair in the injured central nervous system: Lessons from neuronal development.Systemic or Forebrain Neuron-Specific Deficiency of Geranylgeranyltransferase-1 Impairs Synaptic Plasticity and Reduces Dendritic Spine Density.Isoprenoids and protein prenylation: implications in the pathogenesis and therapeutic intervention of Alzheimer's disease.Scaffold Ranking and Positional Scanning Identify Novel Neurite Outgrowth Promoters with Nanomolar Potency
P2860
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P2860
Protein Prenylation Constitutes an Endogenous Brake on Axonal Growth.
description
2016 nî lūn-bûn
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2016年の論文
@ja
2016年学术文章
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2016年学术文章
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2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
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2016年学术文章
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2016年學術文章
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2016年學術文章
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name
Protein Prenylation Constitutes an Endogenous Brake on Axonal Growth.
@en
Protein Prenylation Constitutes an Endogenous Brake on Axonal Growth.
@nl
type
label
Protein Prenylation Constitutes an Endogenous Brake on Axonal Growth.
@en
Protein Prenylation Constitutes an Endogenous Brake on Axonal Growth.
@nl
prefLabel
Protein Prenylation Constitutes an Endogenous Brake on Axonal Growth.
@en
Protein Prenylation Constitutes an Endogenous Brake on Axonal Growth.
@nl
P2093
P50
P1433
P1476
Protein Prenylation Constitutes an Endogenous Brake on Axonal Growth.
@en
P2093
Brent Stockwell
Christopher E Henderson
Derek Oakley
Elena Nikulina
Emily Rhodes Lowry
Gist F Croft
Hynek Wichterle
Jianwei Hou
Marie T Filbin
P304
P356
10.1016/J.CELREP.2016.06.013
P577
2016-06-29T00:00:00Z