Complement C5a potentiates uric acid crystal-induced IL-1β production. - Wikidata - awgldk - TriplyDB

Complement C5a potentiates uric acid crystal-induced IL-1β production.

Complement C5a potentiates uric acid crystal-induced IL-1β production.

http://www.wikidata.org/entity/Q51697175

about

Neutrophil microvesicles resolve gout by inhibiting C5a-mediated priming of the inflammasome Inflammation and its role in age-related macular degeneration Drivers of age-related inflammation and strategies for healthspan extension Complementing the inflammasome.K+ efflux agonists induce NLRP3 inflammasome activation independently of Ca2+ signaling.Editorial: Can GPR43 Sensing of Short-Chain Fatty Acids Unchain Inflammasome-Driven Arthritis?C5a Induces the Synthesis of IL-6 and TNF-α in Rat Glomerular Mesangial Cells through MAPK Signaling Pathways.Complement-mediated 'bystander' damage initiates host NLRP3 inflammasome activation.Bidirectional Crosstalk between C5a Receptors and the NLRP3 Inflammasome in Macrophages and Monocytes.A novel "complement-metabolism-inflammasome axis" as a key regulator of immune cell effector function Monosodium urate crystal-induced pro-interleukin-1β production is post-transcriptionally regulated via the p38 signaling pathway in human monocytes.Serum Protein KNG1, APOC3, and PON1 as Potential Biomarkers for Yin-Deficiency-Heat Syndrome.C5a Regulates IL-1β Production and Leukocyte Recruitment in a Murine Model of Monosodium Urate Crystal-Induced Peritonitis.Synovial fluid proteins are required for the induction of interleukin-1β production by monosodium urate crystals.Innate immune memory: Implications for host responses to damage-associated molecular patterns.Complement Component C5a Primes Retinal Pigment Epithelial Cells for Inflammasome Activation by Lipofuscin-mediated Photooxidative Damage Review: Extracellular Vesicles in Joint Inflammation.Complement in Kidney Transplantation Complement C5a induces PD-L1 expression and acts in synergy with LPS through Erk1/2 and JNK signaling pathways.Inflammation in gout: mechanisms and therapeutic targets.Macrophage Recognition of Crystals and Nanoparticles.Placental inflammation in pre-eclampsia by Nod-like receptor protein (NLRP)3 inflammasome activation in trophoblasts.

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P2860

Complement C5a potentiates uric acid crystal-induced IL-1β production.

http://www.wikidata.org/entity/Q51697175

description

2014 nî lūn-bûn

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2014年の論文

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年學術文章

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2014年學術文章

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name

Complement C5a potentiates uric acid crystal-induced IL-1β production.

@en

Complement C5a potentiates uric acid crystal-induced IL-1β production.

@nl

type

label

Complement C5a potentiates uric acid crystal-induced IL-1β production.

@en

Complement C5a potentiates uric acid crystal-induced IL-1β production.

@nl

prefLabel

Complement C5a potentiates uric acid crystal-induced IL-1β production.

@en

Complement C5a potentiates uric acid crystal-induced IL-1β production.

@nl

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European Journal of Immunology

P1476

Complement C5a potentiates uric acid crystal-induced IL-1β production.

@en

P2093

Brian Naiman

http://www.w3.org/2001/XMLSchema#string

Jane Connor

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Linda Xu

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Ling-Ling An

http://www.w3.org/2001/XMLSchema#string

Michael Fung

http://www.w3.org/2001/XMLSchema#string

Miguel Sanjuan

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Payal Mehta

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Roland Kolbeck

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Sean Turman

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Thornik Reimer

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P2860

NLRP3 is activated in Alzheimer's disease and contributes to pathology in APP/PS1 mice

Gout-associated uric acid crystals activate the NALP3 inflammasome

The dark side of C5a in sepsis

Characterization of a functional NF-kappa B site in the human interleukin 1 beta promoter: evidence for a positive autoregulatory loop

The NALP3 inflammasome is involved in the innate immune response to amyloid-beta

NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals

Fatty acid-induced NLRP3-ASC inflammasome activation interferes with insulin signaling

The NLRP3 inflammasome instigates obesity-induced inflammation and insulin resistance

The inflammasomes: guardians of the body

K⁺ efflux is the common trigger of NLRP3 inflammasome activation by bacterial toxins and particulate matter

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3669-3679

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10.1002/EJI.201444560

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12

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44

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25229885

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