Tacrolimus Reverses UVB Irradiation-Induced Epidermal Langerhans Cell Reduction by Inhibiting TNF-α Secretion in Keratinocytes via Regulation of NF-κB/p65.

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Tacrolimus Reverses UVB Irradiation-Induced Epidermal Langerhans Cell Reduction by Inhibiting TNF-α Secretion in Keratinocytes via Regulation of NF-κB/p65.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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Tacrolimus Reverses UVB Irradi ...... s via Regulation of NF-κB/p65.

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Tacrolimus Reverses UVB Irradi ...... s via Regulation of NF-κB/p65.

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label

Tacrolimus Reverses UVB Irradi ...... s via Regulation of NF-κB/p65.

@en

Tacrolimus Reverses UVB Irradi ...... s via Regulation of NF-κB/p65.

@nl

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Tacrolimus Reverses UVB Irradi ...... s via Regulation of NF-κB/p65.

@en

Tacrolimus Reverses UVB Irradi ...... s via Regulation of NF-κB/p65.

@nl

P1476

Tacrolimus Reverses UVB Irradi ...... s via Regulation of NF-κB/p65.

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P2093

Dan Luo

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GuoXin Song

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JiaLi Xu

http://www.w3.org/2001/XMLSchema#string

Li Yin

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QiXing Gong

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YaDong Feng

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YingYing Hu

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ZhiQiang Yin

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P2860

Interleukin (IL)-18 induces Langerhans cell migration by a tumour necrosis factor-alpha- and IL-1beta-dependent mechanism

Langerhans cells facilitate epithelial DNA damage and squamous cell carcinoma

Dendritic cell migration to lymph nodes: cytokines, chemokines, and lipid mediators.

Topical pimecrolimus inhibits high-dose UVB irradiation-induced epidermal Langerhans cell migration, via regulation of TNF-α and E-cadherin.

Depletion of Langerhans cells in human papillomavirus type 16-infected skin is associated with E6-mediated down regulation of E-cadherin.

Systematic review of published trials: long-term safety of topical corticosteroids and topical calcineurin inhibitors in pediatric patients with atopic dermatitis.

Langerhans Cells Facilitate UVB-Induced Epidermal Carcinogenesis.

Skin homing of Langerhans cell precursors: adhesion, chemotaxis, and migration.

Alpha 6 integrins are required for Langerhans cell migration from the epidermis.

Palmitic acid induces production of proinflammatory cytokines interleukin-6, interleukin-1β, and tumor necrosis factor-α via a NF-κB-dependent mechanism in HaCaT keratinocytes.

P304

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P31

scholarly article

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10.3389/FPHAR.2018.00067

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P478

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P577

2018-02-22T00:00:00Z

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P698

29520229

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P921

NF-κB

P932

5827091

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