Disruption of the actin cortex contributes to susceptibility of mammalian cells to nanosecond pulsed electric fields.
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Characterization of Pressure Transients Generated by Nanosecond Electrical Pulse (nsEP) Exposure.Calcium-mediated pore expansion and cell death following nanoelectroporationElectroporation of mammalian cells by nanosecond electric field oscillations and its inhibition by the electric field reversal.Cell Electrosensitization Exists Only in Certain Electroporation BuffersElasticity and tumorigenic characteristics of cells in a monolayer after nanosecond pulsed electric field exposure.nsPEF-induced PIP2 depletion, PLC activity and actin cytoskeletal cortex remodeling are responsible for post-exposure cellular swelling and blebbing.Activation of the phospholipid scramblase TMEM16F by nanosecond pulsed electric fields (nsPEF) facilitates its diverse cytophysiological effects.Electropore Formation in Mechanically Constrained Phospholipid Bilayers.Electrosensitization Increases Antitumor Effectiveness of Nanosecond Pulsed Electric Fields In Vivo.Tubulin's response to external electric fields by molecular dynamics simulations
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P2860
Disruption of the actin cortex contributes to susceptibility of mammalian cells to nanosecond pulsed electric fields.
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Disruption of the actin cortex ...... second pulsed electric fields.
@en
Disruption of the actin cortex ...... second pulsed electric fields.
@nl
type
label
Disruption of the actin cortex ...... second pulsed electric fields.
@en
Disruption of the actin cortex ...... second pulsed electric fields.
@nl
prefLabel
Disruption of the actin cortex ...... second pulsed electric fields.
@en
Disruption of the actin cortex ...... second pulsed electric fields.
@nl
P2093
P2860
P356
P1433
P1476
Disruption of the actin cortex ...... second pulsed electric fields.
@en
P2093
Bennett L Ibey
Caleb Roth
Gary L Thompson
Gleb Tolstykh
Marjorie Kuipers
P2860
P304
P356
10.1002/BEM.21845
P577
2014-02-20T00:00:00Z