Extracellular domain c-kit mutation with duplication of Ser501Ala502 found in gastrointestinal stromal tumors is more imatinib- and nilotinib-sensitive than that with duplication of Ala502Tyr503.

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Extracellular domain c-kit mutation with duplication of Ser501Ala502 found in gastrointestinal stromal tumors is more imatinib- and nilotinib-sensitive than that with duplication of Ala502Tyr503.

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2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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name

Extracellular domain c-kit mut ...... h duplication of Ala502Tyr503.

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Extracellular domain c-kit mut ...... h duplication of Ala502Tyr503.

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Item

label

Extracellular domain c-kit mut ...... h duplication of Ala502Tyr503.

@en

Extracellular domain c-kit mut ...... h duplication of Ala502Tyr503.

@nl

prefLabel

Extracellular domain c-kit mut ...... h duplication of Ala502Tyr503.

@en

Extracellular domain c-kit mut ...... h duplication of Ala502Tyr503.

@nl

P1476

Extracellular domain c-kit mut ...... h duplication of Ala502Tyr503.

@en

P2093

Ikuo Matsuda

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Koji Isozaki

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Mizuka Ohkouchi

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Ning-Ning Liu

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Noriko Kajimoto

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Seiichi Hirota

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Toshirou Nishida

http://www.w3.org/2001/XMLSchema#string

Tsuyoshi Takahashi

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Yasushi Toh

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Yuka Hashikura

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P2860

Gain-of-function mutations of platelet-derived growth factor receptor alpha gene in gastrointestinal stromal tumors.

Kinase mutations and imatinib response in patients with metastatic gastrointestinal stromal tumor.

Gain-of-function mutations of c-kit in human gastrointestinal stromal tumors

Nilotinib in imatinib-resistant CML and Philadelphia chromosome-positive ALL

Efficacy and safety of a specific inhibitor of the BCR-ABL tyrosine kinase in chronic myeloid leukemia

Efficacy and safety of imatinib mesylate in advanced gastrointestinal stromal tumors

Disturbed intestinal movement, bile reflux to the stomach, and deficiency of c-kit-expressing cells in Ws/Ws mutant rats.

Comparison of two doses of imatinib for the treatment of unresectable or metastatic gastrointestinal stromal tumors: a meta-analysis of 1,640 patients.

PDGFRA activating mutations in gastrointestinal stromal tumors.

A new mutation in the KIT ATP pocket causes acquired resistance to imatinib in a gastrointestinal stromal tumor patient.

P2888

labinvest.2013.43

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502-507

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scholarly article

P356

10.1038/LABINVEST.2013.43

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P433

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P478

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P577

2013-03-04T00:00:00Z

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P5875

235786664

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P698

23459373

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