Selective cyclo-oxygenase-2 inhibition induces regression of autologous endometrial grafts by down-regulation of vascular endothelial growth factor-mediated angiogenesis and stimulation of caspase-3-dependent apoptosis.
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Recent advances in the understanding of endometriosis: the role of inflammatory mediators in disease pathogenesis and treatmentVasculogenesis: a new piece of the endometriosis puzzleThe role of angiogenic factors in fibroid pathogenesis: potential implications for future therapyExpansion of microvascular networks in vivo by phthalimide neovascular factor 1 (PNF1)Selective inhibition of prostaglandin E2 receptors EP2 and EP4 inhibits adhesion of human endometriotic epithelial and stromal cells through suppression of integrin-mediated mechanismsHigh-resolution ultrasound imaging: a novel technique for the noninvasive in vivo analysis of endometriotic lesion and cyst formation in small animal models.Selective inhibition of prostaglandin E2 receptors EP2 and EP4 induces apoptosis of human endometriotic cells through suppression of ERK1/2, AKT, NFkappaB, and beta-catenin pathways and activation of intrinsic apoptotic mechanisms.Anti-angiogenic treatment strategies for the therapy of endometriosis.In vitro and in vivo evaluation of the anti-angiogenic actions of 4-hydroxybenzyl alcohol.A rare case of endometrioma in a bitch.Molecular and preclinical basis to inhibit PGE2 receptors EP2 and EP4 as a novel nonsteroidal therapy for endometriosis.Inhibition of Hyaluronic Acid Synthesis Suppresses Angiogenesis in Developing Endometriotic Lesions.Euterpe oleracea Extract (Açaí) Is a Promising Novel Pharmacological Therapeutic Treatment for Experimental Endometriosis.DNA hypomethylation of the COX-2 gene promoter is associated with up-regulation of its mRNA expression in eutopic endometrium of endometriosis.In vitro and in vivo approaches to study angiogenesis in the pathophysiology and therapy of endometriosis.Deficiency of immunophilin FKBP52 promotes endometriosis.The role of microRNAs in endometriosis and associated reproductive conditions.Prostaglandin E2: the master of endometriosis?Aberrant expression of apoptosis-related molecules in endometriosis: a possible mechanism underlying the pathogenesis of endometriosis.The translational challenge in the development of new and effective therapies for endometriosis: a review of confidence from published preclinical efficacy studies.Techniques for visualization of cell-cell contact at the fetal-maternal interface.Pyrrolidine dithiocarbamate attenuates nuclear factor-ĸB activation, cyclooxygenase-2 expression and prostaglandin E2 production in human endometriotic epithelial cells.MAP kinases and the inflammatory signaling cascade as targets for the treatment of endometriosis?The Effect of Early Postoperative Non-Steroidal Anti-Inflammatory Drugs on Pancreatic Fistula Following Pancreaticoduodenectomy.Effect of dienogest administration on angiogenesis and hemodynamics in a rat endometrial autograft model.Genistein does not affect vascularization and blood perfusion of endometriotic lesions and ovarian follicles in dorsal skinfold chambers of Syrian golden hamsters.Hypoxia-inhibited dual-specificity phosphatase-2 expression in endometriotic cells regulates cyclooxygenase-2 expression.Effects of celecoxib and nimesulide on the proliferation of ectopic endometrial stromal cells in vitro.Concentration effect of prostaglandin E2 on the growth factor expression and cell proliferation in bovine endometrial explants and their kinetic characteristics.Targeting hypoxia-mediated YAP1 nuclear translocation ameliorates pathogenesis of endometriosis without compromising maternal fertility.Combination therapy with telmisartan and parecoxib induces regression of endometriotic lesions.Comparison between resveratrol and cabergoline in preventing ovarian hyperstimulation syndrome in a rat model.Combined blockade of angiotensin II type 1 receptor and activation of peroxisome proliferator-activated receptor-γ by telmisartan effectively inhibits vascularization and growth of murine endometriosis-like lesions.6-Shogaol reduces progression of experimental endometriosis and regulation of VGEF and inhibition of COX-2 and PGE2-mediated inflammatory responsesNew drugs in development for the treatment of endometriosis
P2860
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P2860
Selective cyclo-oxygenase-2 inhibition induces regression of autologous endometrial grafts by down-regulation of vascular endothelial growth factor-mediated angiogenesis and stimulation of caspase-3-dependent apoptosis.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh-hant
name
Selective cyclo-oxygenase-2 in ...... caspase-3-dependent apoptosis.
@en
Selective cyclo-oxygenase-2 in ...... caspase-3-dependent apoptosis.
@nl
type
label
Selective cyclo-oxygenase-2 in ...... caspase-3-dependent apoptosis.
@en
Selective cyclo-oxygenase-2 in ...... caspase-3-dependent apoptosis.
@nl
prefLabel
Selective cyclo-oxygenase-2 in ...... caspase-3-dependent apoptosis.
@en
Selective cyclo-oxygenase-2 in ...... caspase-3-dependent apoptosis.
@nl
P2093
P1476
Selective cyclo-oxygenase-2 in ...... caspase-3-dependent apoptosis.
@en
P2093
Antje Elitzsch
Brigitte Vollmar
Claudia Scheuer
Matthias W Laschke
Michael D Menger
P304
P356
10.1016/J.FERTNSTERT.2006.05.068
P407
P577
2006-11-01T00:00:00Z