about
Induced pluripotent stem cell modeling of multisystemic, hereditary transthyretin amyloidosis.Identification of Transthyretin Cardiac Amyloidosis Using Serum Retinol-Binding Protein 4 and a Clinical Prediction Model.Inhibition of the NOD-Like Receptor Protein 3 Inflammasome Is Protective in Juvenile Influenza A Virus Infection.Serum Proteomic Variability Associated with Clinical Phenotype in Familial Transthyretin Amyloidosis (ATTRm).Blood Proteomic Profiling in Inherited (ATTRm) and Acquired (ATTRwt) Forms of Transthyretin-Associated Cardiac Amyloidosis.Use of Serum Transthyretin as a Prognostic Indicator and Predictor of Outcome in Cardiac Amyloid Disease Associated With Wild-Type Transthyretin.Retinol binding protein 4 (RBP4) concentration identifies V122I transthyretin cardiac amyloidosis.Lung Interstitial Macrophages: Establishing Identity and Uncovering Heterogeneity.Inflammatory Monocytes Drive Influenza A Virus-Mediated Lung Injury in Juvenile Mice.
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description
hulumtuese
@sq
onderzoeker
@nl
researcher
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հետազոտող
@hy
name
Clarissa M Koch
@ast
Clarissa M Koch
@en
Clarissa M Koch
@es
Clarissa M Koch
@nl
Clarissa M Koch
@sl
type
label
Clarissa M Koch
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Clarissa M Koch
@en
Clarissa M Koch
@es
Clarissa M Koch
@nl
Clarissa M Koch
@sl
prefLabel
Clarissa M Koch
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Clarissa M Koch
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Clarissa M Koch
@es
Clarissa M Koch
@nl
Clarissa M Koch
@sl
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0000-0002-7424-6009