Spontaneous action potential generation due to persistent sodium channel currents in simulated carotid body afferent fibers.
about
A painful neuropathy-associated Nav1.7 mutant leads to time-dependent degeneration of small-diameter axons associated with intracellular Ca2+ dysregulation and decrease in ATP levels.Sodium-calcium exchanger and multiple sodium channel isoforms in intra-epidermal nerve terminals.Neuropathy-associated Nav1.7 variant I228M impairs integrity of dorsal root ganglion neuron axons.Membrane properties and electrogenesis in the distal axons of small dorsal root ganglion neurons in vitro.Small nerve fibres, small hands and small feet: a new syndrome of pain, dysautonomia and acromesomelia in a kindred with a novel NaV1.7 mutation.
P2860
Spontaneous action potential generation due to persistent sodium channel currents in simulated carotid body afferent fibers.
description
2008 nî lūn-bûn
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2008年の論文
@ja
2008年学术文章
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2008年学术文章
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2008年学术文章
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2008年学术文章
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2008年學術文章
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name
Spontaneous action potential g ...... carotid body afferent fibers.
@en
Spontaneous action potential g ...... carotid body afferent fibers.
@nl
type
label
Spontaneous action potential g ...... carotid body afferent fibers.
@en
Spontaneous action potential g ...... carotid body afferent fibers.
@nl
prefLabel
Spontaneous action potential g ...... carotid body afferent fibers.
@en
Spontaneous action potential g ...... carotid body afferent fibers.
@nl
P2860
P1476
Spontaneous action potential g ...... carotid body afferent fibers.
@en
P2093
David F Donnelly
P2860
P304
P356
10.1152/JAPPLPHYSIOL.01169.2007
P407
P577
2008-02-28T00:00:00Z