CCR7 ligands CCL19 and CCL21 increase permissiveness of resting memory CD4+ T cells to HIV-1 infection: a novel model of HIV-1 latency.
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How does the timing of antiretroviral therapy initiation in acute infection affect HIV reservoirs?Redefining the viral reservoirs that prevent HIV-1 eradicationThe immunological synapse: the gateway to the HIV reservoirRecent developments in human immunodeficiency virus-1 latency researchHuman immunodeficiency virus (HIV)-1 integration sites in viral latencyUnderstanding HIV latency: the road to an HIV cureAn in-depth comparison of latent HIV-1 reactivation in multiple cell model systems and resting CD4+ T cells from aviremic patientsDirectly infected resting CD4+T cells can produce HIV Gag without spreading infection in a model of HIV latencyThe CCR5-antagonist Maraviroc reverses HIV-1 latency in vitro alone or in combination with the PKC-agonist Bryostatin-1HIV persistence: chemokines and their signalling pathways.Entinostat is a histone deacetylase inhibitor selective for class 1 histone deacetylases and activates HIV production from latently infected primary T cells.Quantitative phosphoproteomics of CXCL12 (SDF-1) signaling.Establishment of HIV-1 latency in resting CD4+ T cells depends on chemokine-induced changes in the actin cytoskeleton.Immune activation and HIV persistence: implications for curative approaches to HIV infection.Finding a cure for HIV: will it ever be achievable?Towards an HIV cure: a global scientific strategyDendritic cell-induced activation of latent HIV-1 provirus in actively proliferating primary T lymphocytesInduction of indoleamine 2,3-dioxygenase by Borrelia burgdorferi in human immune cells correlates with pathogenic potential.Translational challenges in targeting latent HIV infection and the CNS reservoir problem.Myeloid dendritic cells induce HIV-1 latency in non-proliferating CD4+ T cells.Increased expression of the homeostatic chemokines CCL19 and CCL21 in clinical and experimental Rickettsia conorii infectionA primary CD4(+) T cell model of HIV-1 latency established after activation through the T cell receptor and subsequent return to quiescenceEradicating HIV-1 infection: seeking to clear a persistent pathogen.Spinoculation triggers dynamic actin and cofilin activity that facilitates HIV-1 infection of transformed and resting CD4 T cells.Expression and reactivation of HIV in a chemokine induced model of HIV latency in primary resting CD4+ T cells.HIV Latency Is Established Directly and Early in Both Resting and Activated Primary CD4 T Cells.Cannabinoid receptor 2-mediated attenuation of CXCR4-tropic HIV infection in primary CD4+ T cellsH3K27 Demethylation at the Proviral Promoter Sensitizes Latent HIV to the Effects of Vorinostat in Ex Vivo Cultures of Resting CD4+ T Cells.The role of antigen presenting cells in the induction of HIV-1 latency in resting CD4(+) T-cells.Understanding Factors That Modulate the Establishment of HIV Latency in Resting CD4+ T-Cells In Vitro.HIV persistence in the setting of antiretroviral therapy: when, where and how does HIV hide?Temporal transcriptional response to latency reversing agents identifies specific factors regulating HIV-1 viral transcriptional switchLatent HIV-1 infection occurs in multiple subsets of hematopoietic progenitor cells and is reversed by NF-κB activation.The trinity of the cortical actin in the initiation of HIV-1 infectionA new way of measuring apoptosis by absolute quantitation of inter-nucleosomally fragmented genomic DNA.Latent HIV-1 is activated by exosomes from cells infected with either replication-competent or defective HIV-1.Activation and lysis of human CD4 cells latently infected with HIV-1Involvement of LIM kinase 1 in actin polarization in human CD4 T cells.A dichotomy in cortical actin and chemotactic actin activity between human memory and naive T cells contributes to their differential susceptibility to HIV-1 infectionHuman immunodeficiency virus (HIV) latency: the major hurdle in HIV eradication
P2860
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P2860
CCR7 ligands CCL19 and CCL21 increase permissiveness of resting memory CD4+ T cells to HIV-1 infection: a novel model of HIV-1 latency.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
CCR7 ligands CCL19 and CCL21 i ...... novel model of HIV-1 latency.
@en
CCR7 ligands CCL19 and CCL21 i ...... novel model of HIV-1 latency.
@nl
type
label
CCR7 ligands CCL19 and CCL21 i ...... novel model of HIV-1 latency.
@en
CCR7 ligands CCL19 and CCL21 i ...... novel model of HIV-1 latency.
@nl
prefLabel
CCR7 ligands CCL19 and CCL21 i ...... novel model of HIV-1 latency.
@en
CCR7 ligands CCL19 and CCL21 i ...... novel model of HIV-1 latency.
@nl
P2093
P1433
P1476
CCR7 ligands CCL19 and CCL21 i ...... novel model of HIV-1 latency.
@en
P2093
Ajantha Solomon
Miranda Xhilaga
Paul U Cameron
Suha Saleh
P304
P356
10.1182/BLOOD-2007-06-097907
P407
P577
2007-09-19T00:00:00Z