Increased Toll-like receptor 4 expression on T cells may be a mechanism for enhanced T cell response late after burn injury.
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New vaccines against influenza virus.Up-regulation of cell surface Toll-like receptors on circulating gammadelta T-cells following burn injury.The TLR3 agonist poly(I:C) targets CD8+ T cells and augments their antigen-specific responses upon their adoptive transfer into naïve recipient miceTranscriptional analysis of infiltrating T cells in kidney ischemia-reperfusion injury reveals a pathophysiological role for CCR5.Burn enhances toll-like receptor induced responses by circulating leukocytes.Dermal γδ T-Cells Can Be Activated by Mitochondrial Damage-Associated Molecular Patterns.Burkholderia pseudomallei infection of T cells leads to T-cell costimulation partially provided by flagellin.Increased serum NKG2D-ligands and downregulation of NKG2D in peripheral blood NK cells of patients with major burns.The molecular mechanisms of action of PPAR-γ agonists in the treatment of corneal alkali burns (Review)Flagellin treatment prevents increased susceptibility to systemic bacterial infection after injury by inhibiting anti-inflammatory IL-10+ IL-12- neutrophil polarization.The lncRNAs involved in mouse airway allergic inflammation following induced pluripotent stem cell-mesenchymal stem cell treatment.Endogenous toll-like receptor ligands and their biological significance.Insights into the Relationship between Toll Like Receptors and Gamma Delta T Cell Responses.Timeline of health care-associated infections and pathogens after burn injuries.Signaling through the adaptor molecule MyD88 in CD4+ T cells is required to overcome suppression by regulatory T cells.Innate and adaptive immunity gene expression of human keratinocytes cultured of severe burn injury.Burn-injury affects gut-associated lymphoid tissues derived CD4+ T cells.OX62+OX6+OX35+ rat dendritic cells are unable to prime CD4+ T cells for an effective immune response following acute burn injury.Significant correlation of TLR4 expression with the clinicopathological features of invasive ductal carcinoma of the breast.Blocking CXCL1-dependent neutrophil recruitment prevents immune damage and reduces pulmonary bacterial infection after inhalation injury.Risk Factors for Healthcare-Associated Infections in Adult Burn Patients.Toll-like receptors: Significance, ligands, signaling pathways, and functions in mammals.Damage-associated molecular patterns (DAMPs) released after burn are associated with inflammation and monocyte activation.
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P2860
Increased Toll-like receptor 4 expression on T cells may be a mechanism for enhanced T cell response late after burn injury.
description
2006 nî lūn-bûn
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name
Increased Toll-like receptor 4 ...... sponse late after burn injury.
@en
Increased Toll-like receptor 4 ...... sponse late after burn injury.
@nl
type
label
Increased Toll-like receptor 4 ...... sponse late after burn injury.
@en
Increased Toll-like receptor 4 ...... sponse late after burn injury.
@nl
prefLabel
Increased Toll-like receptor 4 ...... sponse late after burn injury.
@en
Increased Toll-like receptor 4 ...... sponse late after burn injury.
@nl
P2093
P1433
P1476
Increased Toll-like receptor 4 ...... sponse late after burn injury.
@en
P2093
Anthony A Meyer
Bruce Cairns
Carie M Barnes
Jeffrey A Frelinger
P304
293-8; discussion 298-9
P356
10.1097/01.TA.0000228969.46633.BB
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P50
P577
2006-08-01T00:00:00Z