Folate supplementation during pregnancy improves offspring cardiovascular dysfunction induced by protein restriction.
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Maternal Micronutrient Deficiency, Fetal Development, and the Risk of Chronic DiseaseThe Programming Power of the PlacentaFetal stress and programming of hypoxic/ischemic-sensitive phenotype in the neonatal brain: mechanisms and possible interventionsGene-environment interactions in severe intraventricular hemorrhage of preterm neonatesEpigenetic modifications: basic mechanisms and role in cardiovascular disease.Chronic maternal vitamin B12 restriction induced changes in body composition & glucose metabolism in the Wistar rat offspring are partly correctable by rehabilitationEffect of hyperglycaemia in pregnancy on adiposity in their infants in India: a protocol of a multicentre cohort studyEarly origins of adult disease: approaches for investigating the programmable epigenome in humans, nonhuman primates, and rodentsGene expression in the placenta: maternal stress and epigenetic responses.Embryo transfer cannot delineate between the maternal pregnancy environment and germ line effects in the transgenerational transmission of disease in rats.Maternal obesity, inflammation, and developmental programming.Experimental intrauterine growth restriction induces alterations in DNA methylation and gene expression in pancreatic islets of rats.An evolving scientific basis for the prevention and treatment of pediatric obesityTaurine supplementation reduces blood pressure and prevents endothelial dysfunction and oxidative stress in post-weaning protein-restricted rats.Vascular dysfunction induced in offspring by maternal dietary fat involves altered arterial polyunsaturated fatty acid biosynthesisPre-weaning growth hormone treatment reverses hypertension and endothelial dysfunction in adult male offspring of mothers undernourished during pregnancy.Induction of altered epigenetic regulation of the hepatic glucocorticoid receptor in the offspring of rats fed a protein-restricted diet during pregnancy suggests that reduced DNA methyltransferase-1 expression is involved in impaired DNA methylatioGestational hypoxia and epigenetic programming of brain development disorders.Dietary alleviation of maternal obesity and diabetes: increased resistance to diet-induced obesity transcriptional and epigenetic signatures.Excess of methyl donor in the perinatal period reduces postnatal leptin secretion in rat and interacts with the effect of protein content in dietMechanisms of developmental programming of the metabolic syndrome and related disordersDiet, nutrition and modulation of genomic expression in fetal origins of adult diseaseDevelopmental programming of cardiovascular disease following intrauterine growth restriction: findings utilising a rat model of maternal protein restriction.Genome-wide methylation and gene expression changes in newborn rats following maternal protein restriction and reversal by folic acidDevelopmental programming of the metabolic syndrome - critical windows for intervention.Epigenetic mechanisms in developmental programming of adult disease.Appetite regulation is independent of the changes in ghrelin levels in pregnant rats fed low-protein dietEndothelial Functioning and Hemodynamic Parameters in Rats with Subclinical Hypothyroid and the Effects of Thyroxine ReplacementMismatched pre- and postnatal nutrition leads to cardiovascular dysfunction and altered renal function in adulthood.Molecular mechanisms underlying the fetal programming of adult disease.Enalapril Normalizes Endothelium-Derived Hyperpolarizing Factor-Mediated Relaxation in Mesenteric Artery of Adult Hypertensive Rats Prenatally Exposed to Testosterone.Epigenetic mechanisms and the mismatch concept of the developmental origins of health and disease.The nature of the growth pattern and of the metabolic response to fasting in the rat are dependent upon the dietary protein and folic acid intakes of their pregnant dams and post-weaning fat consumption.Recent progress toward the understanding of the pathophysiology of hypertension during preeclampsia.Nutrition in early life, and risk of cancer and metabolic disease: alternative endings in an epigenetic tale?Effects of multivitamins and known teratogens on chick cardiomyocytes micromass culture assay.Nutritional programming of disease: unravelling the mechanism.Blastocyst environment and its influence on offspring cardiovascular health: the heart of the matter.Developmental programming and hypertension.Epigenetics in cardiovascular disease
P2860
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P2860
Folate supplementation during pregnancy improves offspring cardiovascular dysfunction induced by protein restriction.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
Folate supplementation during ...... nduced by protein restriction.
@en
Folate supplementation during ...... nduced by protein restriction.
@nl
type
label
Folate supplementation during ...... nduced by protein restriction.
@en
Folate supplementation during ...... nduced by protein restriction.
@nl
prefLabel
Folate supplementation during ...... nduced by protein restriction.
@en
Folate supplementation during ...... nduced by protein restriction.
@nl
P2093
P50
P1433
P1476
Folate supplementation during ...... nduced by protein restriction.
@en
P2093
Alan A Jackson
Caroline S Dance
Frederick W Anthony
Lee Brawley
Rebecca Dunn
P304
P356
10.1161/01.HYP.0000215580.43711.D1
P407
P577
2006-04-03T00:00:00Z