Vascularization of the mouse embryo: a study of flk-1, tek, tie, and vascular endothelial growth factor expression during development.
about
Multiple angiopoietin recombinant proteins activate the Tie1 receptor tyrosine kinase and promote its interaction with Tie2CYR61 (CCN1) is essential for placental development and vascular integrityVE-statin, an endothelial repressor of smooth muscle cell migrationThe vascular endothelial growth factor (VEGF) family: angiogenic factors in health and diseaseEmbryogenesis of the first circulating endothelial cellsRescue of the early vascular defects in Tek/Tie2 null mice reveals an essential survival functionQuercetin-4'-O-β-D-glucopyranoside (QODG) inhibits angiogenesis by suppressing VEGFR2-mediated signaling in zebrafish and endothelial cellsVascular endothelial growth factor A (VEGF-A) is involved in guidance of VEGF receptor-positive cells to the anterior portion of early embryosFormation and differentiation of multiple mesenchymal lineages during lung development is regulated by beta-catenin signalingProtocadherin 12 deficiency alters morphogenesis and transcriptional profile of the placentaInhibition of tumor angiogenesis using a soluble receptor establishes a role for Tie2 in pathologic vascular growthAmphetamine and environmentally induced hyperthermia differentially alter the expression of genes regulating vascular tone and angiogenesis in the meninges and associated vasculatureProgressive loss of the spongiotrophoblast layer of Birc6/Bruce mutants results in embryonic lethalityER71 acts downstream of BMP, Notch, and Wnt signaling in blood and vessel progenitor specificationOtt1 (Rbm15) is essential for placental vascular branching morphogenesis and embryonic development of the heart and spleenCombinatorial effects of Flk1 and Tal1 on vascular and hematopoietic development in the mouseEssential role for p38alpha mitogen-activated protein kinase in placental angiogenesisBRUCE, a giant E2/E3 ubiquitin ligase and inhibitor of apoptosis protein of the trans-Golgi network, is required for normal placenta development and mouse survivalTyrosine 1101 of Tie2 is the major site of association of p85 and is required for activation of phosphatidylinositol 3-kinase and AktIdentification of progenitor cells that contribute to heterotopic skeletogenesisZebrafish Tie-2 shares a redundant role with Tie-1 in heart development and regulates vessel integrity.Expression and function of angiopoietin-1 in breast cancer.Determination of hypoxic region by hypoxia marker in developing mouse embryos in vivo: a possible signal for vessel development.Effect of angiogenic and antiangiogenic compounds on the outgrowth of capillary structures from fetal mouse bone explants.Vascular endothelial-specific dimethylarginine dimethylaminohydrolase-1-deficient mice reveal that vascular endothelium plays an important role in removing asymmetric dimethylarginine.Segregation of the embryonic vascular and hemopoietic systems.The clinical manipulation of angiogenesis: pathology, side-effects, surprises, and opportunities with novel human therapies.The vascular endothelial growth factor family: signalling for vascular development.Transgenic mouse models in angiogenesis and cardiovascular disease.Placental failure in mice lacking the mammalian homolog of glial cells missing, GCMa.Early embryonic vascular patterning by matrix-mediated paracrine signalling: a mathematical model study.Angiogenesis in the placenta.Transcription factors in mouse lung development and function.Hypoxia-inducible factor and the development of stem cells of the cardiovascular system.The endothelial receptor tyrosine kinase Tie1 activates phosphatidylinositol 3-kinase and Akt to inhibit apoptosis.Hypoxia influences the vascular expansion and differentiation of embryonic stem cell cultures through the temporal expression of vascular endothelial growth factor receptors in an ARNT-dependent manner.Pathways of cell activation in spondyloarthropathies.Role of hematopoietic stem cells in angiogenesis.Active adenoviral vascular penetration by targeted formation of heterocellular endothelial-epithelial syncytia.Potentiation of angiogenic response by ischemic and hypoxic preconditioning of the heart.
P2860
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P2860
Vascularization of the mouse embryo: a study of flk-1, tek, tie, and vascular endothelial growth factor expression during development.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh-hant
name
Vascularization of the mouse e ...... expression during development.
@en
Vascularization of the mouse e ...... expression during development.
@nl
type
label
Vascularization of the mouse e ...... expression during development.
@en
Vascularization of the mouse e ...... expression during development.
@nl
prefLabel
Vascularization of the mouse e ...... expression during development.
@en
Vascularization of the mouse e ...... expression during development.
@nl
P2093
P356
P1476
Vascularization of the mouse e ...... expression during development.
@en
P2093
P356
10.1002/AJA.1002030109
P577
1995-05-01T00:00:00Z