MALAT1: An Epigenetic Regulator of Inflammation in Diabetic Retinopathy.

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MALAT1: An Epigenetic Regulator of Inflammation in Diabetic Retinopathy.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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MALAT1: An Epigenetic Regulator of Inflammation in Diabetic Retinopathy.

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MALAT1: An Epigenetic Regulator of Inflammation in Diabetic Retinopathy.

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MALAT1: An Epigenetic Regulator of Inflammation in Diabetic Retinopathy.

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MALAT1: An Epigenetic Regulator of Inflammation in Diabetic Retinopathy.

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MALAT1: An Epigenetic Regulator of Inflammation in Diabetic Retinopathy.

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MALAT1: An Epigenetic Regulator of Inflammation in Diabetic Retinopathy.

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P1476

MALAT1: An Epigenetic Regulator of Inflammation in Diabetic Retinopathy.

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P2093

Anu Alice Thomas

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Bekim Sadikovic

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Biao Feng

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Erfan Aref-Eshghi

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John Gonder

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Shali Chen

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Subrata Chakrabarti

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P2860

The lncRNA Malat1 is dispensable for mouse development but its transcription plays a cis-regulatory role in the adult

Epigenetic silencing of tumour suppressor gene p15 by its antisense RNA

Transcription of bxd noncoding RNAs promoted by trithorax represses Ubx in cis by transcriptional interference

The human genome browser at UCSC

Targeting histone methyltransferases and demethylases in clinical trials for cancer therapy

RNAi factors are present and active in human cell nuclei

Retinopathy in diabetes

A strategy for probing the function of noncoding RNAs finds a repressor of NFAT

Polycomb proteins targeted by a short repeat RNA to the mouse X chromosome

Molecular mechanisms of long noncoding RNAs

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6526

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scholarly article

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10.1038/S41598-018-24907-W

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English

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P478

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Saumik Biswas

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2018-04-25T00:00:00Z

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29695738

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5916949

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