HMGB1/IL-1β complexes in plasma microvesicles modulate immune responses to burn injury.

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HMGB1/IL-1β complexes in plasma microvesicles modulate immune responses to burn injury.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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HMGB1/IL-1β complexes in plasma microvesicles modulate immune responses to burn injury.

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HMGB1/IL-1β complexes in plasma microvesicles modulate immune responses to burn injury.

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HMGB1/IL-1β complexes in plasma microvesicles modulate immune responses to burn injury.

@en

HMGB1/IL-1β complexes in plasma microvesicles modulate immune responses to burn injury.

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HMGB1/IL-1β complexes in plasma microvesicles modulate immune responses to burn injury.

@en

HMGB1/IL-1β complexes in plasma microvesicles modulate immune responses to burn injury.

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P1476

HMGB1/IL-1β complexes in plasma microvesicles modulate immune responses to burn injury.

@en

P2093

Bruce A Cairns

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Fulton T Crews

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Robert Maile

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Samuel W Jones

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P2860

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The nuclear protein HMGB1 is secreted by monocytes via a non-classical, vesicle-mediated secretory pathway

Platelets amplify inflammation in arthritis via collagen-dependent microparticle production

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HMG-1 as a late mediator of endotoxin lethality in mice

HMGB1 loves company

Establishment and characterization of a human acute monocytic leukemia cell line (THP-1)

HMGB proteins function as universal sentinels for nucleic-acid-mediated innate immune responses

P2X7 receptor differentially couples to distinct release pathways for IL-1beta in mouse macrophage

Nonclassical IL-1 beta secretion stimulated by P2X7 receptors is dependent on inflammasome activation and correlated with exosome release in murine macrophages

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e0195335

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scholarly article

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10.1371/JOURNAL.PONE.0195335

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English

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P478

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Leon G Coleman

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2018-03-30T00:00:00Z

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29601597

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5877880

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