The analysis of new short-period circadian rhythm mutants suggests features of D. melanogaster period gene function.
about
Activating PER repressor through a DBT-directed phosphorylation switchNEMO/NLK phosphorylates PERIOD to initiate a time-delay phosphorylation circuit that sets circadian clock speedTranslational regulation of the DOUBLETIME/CKIδ/ε kinase by LARK contributes to circadian period modulationThe Implications of Multiple Circadian Clock OriginsRhythmic PER abundance defines a critical nodal point for negative feedback within the circadian clock mechanismCircadian transcription contributes to core period determination in Drosophila.Molecular neurogenetics of biological rhythms.timrit Lengthens circadian period in a temperature-dependent manner through suppression of PERIOD protein cycling and nuclear localization.Molecular genetic analysis of circadian timekeeping in Drosophila.Dosage compensation of the period gene in Drosophila melanogaster.Molecular and behavioral analysis of four period mutants in Drosophila melanogaster encompassing extreme short, novel long, and unorthodox arrhythmic typesStoichiometric relationship among clock proteins determines robustness of circadian rhythms.Circadian period integrates network information through activation of the BMP signaling pathway.Temporal phosphorylation of the Drosophila period protein.The phospho-occupancy of an atypical SLIMB-binding site on PERIOD that is phosphorylated by DOUBLETIME controls the pace of the clock.Functional identification of the mouse circadian Clock gene by transgenic BAC rescue.Intergeneric complementation of a circadian rhythmicity defect: phylogenetic conservation of structure and function of the clock gene frequency.Constitutive overexpression of the Drosophila period protein inhibits period mRNA cycling.Masking and the evolution of circadian rhythmicity.NEMO kinase contributes to core period determination by slowing the pace of the Drosophila circadian oscillatorCK1/Doubletime activity delays transcription activation in the circadian clock.Behavior in light-dark cycles of Drosophila mutants that are arrhythmic, blind, or both.The timSL mutant affects a restricted portion of the Drosophila melanogaster circadian cycle.Cis-combination of the classic per(S) and per(L) mutations results in arrhythmic Drosophila with ectopic accumulation of hyperphosphorylated PERIOD protein.
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P2860
The analysis of new short-period circadian rhythm mutants suggests features of D. melanogaster period gene function.
description
1992 nî lūn-bûn
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1992年の論文
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1992年学术文章
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1992年学术文章
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1992年学术文章
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1992年学术文章
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1992年学术文章
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1992年學術文章
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name
The analysis of new short-peri ...... nogaster period gene function.
@en
The analysis of new short-peri ...... nogaster period gene function.
@nl
type
label
The analysis of new short-peri ...... nogaster period gene function.
@en
The analysis of new short-peri ...... nogaster period gene function.
@nl
prefLabel
The analysis of new short-peri ...... nogaster period gene function.
@en
The analysis of new short-peri ...... nogaster period gene function.
@nl
P2093
P2860
P1476
The analysis of new short-peri ...... nogaster period gene function.
@en
P2093
P2860
P304
P356
10.3109/01677069209084155
P577
1992-05-01T00:00:00Z