Gelatinase B is present in the cerebrospinal fluid during experimental autoimmune encephalomyelitis and cleaves myelin basic protein.
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The significance of matrix metalloproteinases in the immunopathogenesis and treatment of multiple sclerosisEndoneurial remodeling by TNFalph- and TNFalpha-releasing proteases. A spatial and temporal co-localization study in painful neuropathy.Matrix metalloproteinase expression and inhibition after sciatic nerve axotomy.The role of TNFalpha and lymphotoxin in demyelinating disease.Cytokine regulation of MMP-9 in peripheral glia: implications for pathological processes and pain in injured nerve.Matrix metalloproteinase inhibition enhances the rate of nerve regeneration in vivo by promoting dedifferentiation and mitosis of supporting schwann cellsMetzincin proteases and their inhibitors: foes or friends in nervous system physiology?Matrix metalloproteinases and neuroinflammation in multiple sclerosis.Matrix metalloproteinase-9 controls proliferation of NG2+ progenitor cells immediately after spinal cord injury.Regulation of matrix metalloproteinases and their inhibitor genes in lipopolysaccharide-induced endotoxemia in mice.Coordinated induction of extracellular proteolysis systems during experimental autoimmune encephalomyelitis in mice.Differential expression of matrix metalloproteinase and tissue inhibitor of matrix metalloproteinase genes in the mouse central nervous system in normal and inflammatory states.Dynamics of matrix turnover during pathologic remodeling of the extracellular matrix.Reversal of experimental autoimmune encephalomyelitis with a hydroxamate inhibitor of matrix metalloproteasesSpinal matrix metalloproteinase 3 mediates inflammatory hyperalgesia via a tumor necrosis factor-dependent mechanism.The axon-glia unit in white matter stroke: mechanisms of damage and recovery.Rescue from acute neuroinflammation by pharmacological chemokine-mediated deviation of leukocytes.Cleavage of myelin associated glycoprotein by matrix metalloproteinases.Divergent role for MMP-2 in myelin breakdown and oligodendrocyte death following transient global ischemia.Matrix metalloproteinase-2 facilitates wound healing events that promote functional recovery after spinal cord injury.A targeted DNA vaccine encoding fas ligand defines its dual role in the regulation of experimental autoimmune encephalomyelitisBowman-Birk inhibitor suppresses autoimmune inflammation and neuronal loss in a mouse model of multiple sclerosis.Matrix metalloproteinases limit functional recovery after spinal cord injury by modulation of early vascular events.The role of microglia and matrix metalloproteinases involvement in neuroinflammation and gliomasInteraction of spirochetes with the host fibrinolytic system and potential roles in pathogenesis.Matrix Metalloproteinases During Axonal Regeneration, a Multifactorial Role from Start to Finish.Reciprocal relationship between membrane type 1 matrix metalloproteinase and the algesic peptides of myelin basic protein contributes to chronic neuropathic pain.Reactivity pattern and cytokine profile of T cells primed by myelin peptides in multiple sclerosis and healthy individuals.Resistance of young gelatinase B-deficient mice to experimental autoimmune encephalomyelitis and necrotizing tail lesions.Matrix metalloproteinase 2 (MMP2) and MMP9 are produced by kidney collecting duct principal cells but are differentially regulated by SV40 large-T, arginine vasopressin, and epidermal growth factor.Cytokines and signaling pathways regulating matrix metalloproteinase-9 (MMP-9) expression in corneal epithelial cells.Dystroglycan is selectively cleaved at the parenchymal basement membrane at sites of leukocyte extravasation in experimental autoimmune encephalomyelitis.Calpain-mediated down-regulation of myelin-associated glycoprotein in lysophosphatidic acid-induced neuropathic painProduction of MMPs in human cerebral endothelial cells and their role in shedding adhesion molecules.Substrate hydrolysis by matrix metalloproteinase-9.Pro-inflammatory cytokines modulate matrix metalloproteinase secretion and organic anion transport at the blood-cerebrospinal fluid barrier.The protease inhibitor, Bowman-Birk Inhibitor, suppresses experimental autoimmune encephalomyelitis: a potential oral therapy for multiple sclerosis.Certain forms of matrix metalloproteinase-9 accumulate in the extracellular space after microdialysis probe implantation and middle cerebral artery occlusion/reperfusion.Up-regulation of MMP-8 and MMP-9 activity in the BALB/c mouse spinal cord correlates with the severity of experimental autoimmune encephalomyelitis.Immunopathology of secondary-progressive multiple sclerosis.
P2860
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P2860
Gelatinase B is present in the cerebrospinal fluid during experimental autoimmune encephalomyelitis and cleaves myelin basic protein.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh-hant
name
Gelatinase B is present in the ...... cleaves myelin basic protein.
@en
Gelatinase B is present in the ...... cleaves myelin basic protein.
@nl
type
label
Gelatinase B is present in the ...... cleaves myelin basic protein.
@en
Gelatinase B is present in the ...... cleaves myelin basic protein.
@nl
prefLabel
Gelatinase B is present in the ...... cleaves myelin basic protein.
@en
Gelatinase B is present in the ...... cleaves myelin basic protein.
@nl
P2093
P356
P1476
Gelatinase B is present in the ...... d cleaves myelin basic protein
@en
P2093
P304
P356
10.1002/JNR.490360409
P577
1993-11-01T00:00:00Z