Disrupted pulmonary vasculature and decreased vascular endothelial growth factor, Flt-1, and TIE-2 in human infants dying with bronchopulmonary dysplasia.
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Maternal Micronutrient Deficiency, Fetal Development, and the Risk of Chronic DiseaseExcess circulating angiopoietin-2 may contribute to pulmonary vascular leak in sepsis in humansAberrant Pulmonary Vascular Growth and Remodeling in Bronchopulmonary DysplasiaPathogenesis of bronchopulmonary dysplasia: when inflammation meets organ developmentBiomarkers for Bronchopulmonary Dysplasia in the Preterm InfantEndogenous and Exogenous Stem/Progenitor Cells in the Lung and Their Role in the Pathogenesis and Treatment of Pediatric Lung DiseaseUnderstanding the Impact of Infection, Inflammation, and Their Persistence in the Pathogenesis of Bronchopulmonary DysplasiaStem cell-based therapies for the newborn lung and brain: Possibilities and challengesStem Cells and Their Mediators - Next Generation Therapy for Bronchopulmonary DysplasiaUtility of large-animal models of BPD: chronically ventilated preterm lambsThe Robyn Barst Memorial Lecture: Differences between the fetal, newborn, and adult pulmonary circulations: relevance for age-specific therapies (2013 Grover Conference series)Role of Nitric Oxide Isoforms in Vascular and Alveolar Development and Lung Injury in Vascular Endothelial Growth Factor Overexpressing Neonatal Mice LungsFrom Here to There, Progenitor Cells and Stem Cells Are Everywhere in Lung Vascular RemodelingHypoxic Episodes in Bronchopulmonary DysplasiaSuppression of inflammatory cell trafficking and alveolar simplification by the heme oxygenase-1 product carbon monoxideDistribution and expression of CD200 in the rat respiratory system under normal and endotoxin-induced pathological conditionsNeonatal oxygen increases sensitivity to influenza A virus infection in adult mice by suppressing epithelial expression of Ear1Mig-6 is required for appropriate lung development and to ensure normal adult lung homeostasisNormal remodeling of the oxygen-injured lung requires the cyclin-dependent kinase inhibitor p21(Cip1/WAF1/Sdi1)Prolonged mechanical ventilation with air induces apoptosis and causes failure of alveolar septation and angiogenesis in lungs of newborn mice.Neonatal lung side population cells demonstrate endothelial potential and are altered in response to hyperoxia-induced lung simplification.Postnatal inflammation in the pathogenesis of bronchopulmonary dysplasiaFinding novel molecular connections between developmental processes and disease.Developmental determinants and changing patterns of respiratory outcomes after preterm birth.The Future of Bronchopulmonary Dysplasia: Emerging Pathophysiological Concepts and Potential New Avenues of Treatment.Soluble platelet-endothelial cell adhesion molecule-1, a biomarker of ventilator-induced lung injuryChronic lung disease in the preterm infant. Lessons learned from animal modelsNeonatal hyperoxia stimulates the expansion of alveolar epithelial type II cellsLung function development after preterm birth in relation to severity of Bronchopulmonary dysplasia.Pulmonary vascular changes in extremely preterm sheep after intra-amniotic exposure to Ureaplasma parvum and lipopolysaccharideActivation of hypoxia-inducible factors in hyperoxia through prolyl 4-hydroxylase blockade in cells and explants of primate lungBronchopulmonary dysplasia: NHLBI Workshop on the Primary Prevention of Chronic Lung DiseasesChronic lung disease in preterm lambs: effect of daily vitamin A treatment on alveolarization.Reduced platelet-derived growth factor receptor expression is a primary feature of human bronchopulmonary dysplasia.Disrupted lung development and bronchopulmonary dysplasia: opportunities for lung repair and regeneration.beta6 Integrin subunit deficiency alleviates lung injury in a mouse model of bronchopulmonary dysplasia.Endoglin in amniotic fluid as a risk factor for the subsequent development of bronchopulmonary dysplasia.Neonatal periostin knockout mice are protected from hyperoxia-induced alveolar simplication.Oxygen differentially affects the hox proteins Hoxb5 and Hoxa5 altering airway branching and lung vascular formation.Cord blood angiogenic progenitor cells are decreased in bronchopulmonary dysplasia.
P2860
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P2860
Disrupted pulmonary vasculature and decreased vascular endothelial growth factor, Flt-1, and TIE-2 in human infants dying with bronchopulmonary dysplasia.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh-hant
name
Disrupted pulmonary vasculatur ...... th bronchopulmonary dysplasia.
@en
Disrupted pulmonary vasculatur ...... th bronchopulmonary dysplasia.
@nl
type
label
Disrupted pulmonary vasculatur ...... th bronchopulmonary dysplasia.
@en
Disrupted pulmonary vasculatur ...... th bronchopulmonary dysplasia.
@nl
prefLabel
Disrupted pulmonary vasculatur ...... th bronchopulmonary dysplasia.
@en
Disrupted pulmonary vasculatur ...... th bronchopulmonary dysplasia.
@nl
P2093
P1476
Disrupted pulmonary vasculatur ...... th bronchopulmonary dysplasia.
@en
P2093
P304
P356
10.1164/AJRCCM.164.10.2101140
P407
P433
P577
2001-11-01T00:00:00Z