Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart. - Wikidata - awgldk - TriplyDB

Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart.

Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart.

http://www.wikidata.org/entity/Q52551784

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A mouse model for Costello syndrome reveals an Ang II-mediated hypertensive condition Scaffold Proteins Regulating Extracellular Regulated Kinase Function in Cardiac Hypertrophy and Disease Gene and cell therapies for the failing heart to prevent sudden arrhythmic death Small G proteins in the cardiovascular system: physiological and pathological aspects Fibroblast growth factor receptor 1 signaling in adult cardiomyocytes increases contractility and results in a hypertrophic cardiomyopathy Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale.Mutations in JPH2-encoded junctophilin-2 associated with hypertrophic cardiomyopathy in humans Oral administration of an angiotensin-converting enzyme 2 activator ameliorates diabetes-induced cardiac dysfunction Gender differences in the modulation of cardiac gene expression by dietary conjugated linoleic acid isomers The ral exchange factor rgl2 promotes cardiomyocyte survival and inhibits cardiac fibrosis Activation of MAPK pathways links LMNA mutations to cardiomyopathy in Emery-Dreifuss muscular dystrophy.Stiffness- and relaxation-based quantitation of radial left ventricular oscillations: elucidation of regional diastolic function mechanisms.Statins and myocardial remodelling: cell and molecular pathways.Crosstalk between mitogen-activated protein kinases and mitochondria in cardiac diseases: therapeutic perspectives MEK-ERK pathway modulation ameliorates disease phenotypes in a mouse model of Noonan syndrome associated with the Raf1(L613V) mutation Capacity for resolution of Ras-MAPK-initiated early pathogenic myocardial hypertrophy modeled in mice.Cyclosporine attenuates cardiomyocyte hypertrophy induced by RAF1 mutants in Noonan and LEOPARD syndromes.SR calcium handling dysfunction, stress-response signaling pathways, and atrial fibrillation.Human Engineered Cardiac Tissues Created Using Induced Pluripotent Stem Cells Reveal Functional Characteristics of BRAF-Mediated Hypertrophic Cardiomyopathy.Quantitative analysis of the chromatin proteome in disease reveals remodeling principles and identifies high mobility group protein B2 as a regulator of hypertrophic growth.The Ca2+ ATPase of cardiac sarcoplasmic reticulum: Physiological role and relevance to diseases Phosphodiesterase 4D and heart failure: a cautionary tale.Mitogen-activated protein kinases in heart development and diseases Regulator of G protein signalling 14 attenuates cardiac remodelling through the MEK-ERK1/2 signalling pathway.Effects of thapsigargin and phenylephrine on calcineurin and protein kinase C signaling functions in cardiac myocytes.Autonomous and Non-autonomous Defects Underlie Hypertrophic Cardiomyopathy in BRAF-Mutant hiPSC-Derived Cardiomyocytes.Cdc42 is an antihypertrophic molecular switch in the mouse heart.Signaling to cardiac hypertrophy: insights from human and mouse RASopathies.Key role of ERK1/2 molecular scaffolds in heart pathology.The Stress-Response MAP Kinase Signaling in Cardiac Arrhythmias.Murine Electrophysiological Models of Cardiac Arrhythmogenesis.H-Ras Isoform Mediates Protection Against Pressure Overload-Induced Cardiac Dysfunction in Part Through Activation of AKT.Cellular interplay via cytokine hierarchy causes pathological cardiac hypertrophy in RAF1-mutant Noonan syndrome.Protein disulfide isomerase increases in myocardial endothelial cells in mice exposed to chronic hypoxia: a stimulatory role in angiogenesis.Phenylephrine hypertrophy, Ca2+-ATPase (SERCA2), and Ca2+ signaling in neonatal rat cardiac myocytes.Pin1 facilitates isoproterenol‑induced cardiac fibrosis and collagen deposition by promoting oxidative stress and activating the MEK1/2‑ERK1/2 signal transduction pathway in rats.Inhibition of mevalonate pathway prevents ischemia-induced cardiac dysfunction in rats via RhoA-independent signaling pathway.Clinical, pathological, and molecular analyses of cardiovascular abnormalities in Costello syndrome: a Ras/MAPK pathway syndrome.Network-based predictions of in vivo cardiac hypertrophy Distinct gene expression profiles in adult mouse heart following targeted MAP kinase activation

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Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart.

http://www.wikidata.org/entity/Q52551784

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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name

Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart.

@en

Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart.

@nl

type

label

Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart.

@en

Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart.

@nl

prefLabel

Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart.

@en

Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart.

@nl

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AJPHEART.00110.2003

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American Journal of Physiology - Heart and Circulatory Physiology

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Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart.

@en

P2093

Jader Dos Santos Cruz

http://www.w3.org/2001/XMLSchema#string

Jeanine A Ursitti

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John Ross

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Jonathan W Lederer

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Ju Chen

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Keith Dilly

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Kenneth R Chien

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Manxiang Li

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Meizi Zheng

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Yusu Gu

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P2860

Selective requirement of myosin light chain 2v in embryonic heart function

Familial hypertrophic cardiomyopathy: from mutations to functional defects

The in vivo role of p38 MAP kinases in cardiac remodeling and restrictive cardiomyopathy

Guanine nucleotide exchange factor-like factor (Rlf) induces gene expression and potentiates alpha 1-adrenergic receptor-induced transcriptional responses in neonatal rat ventricular myocytes.

Protein phosphatase activity is increased in a rat model of long-term beta-adrenergic stimulation.

Ras reduces L-type calcium channel current in cardiac myocytes. Corrective effects of L-channels and SERCA2 on [Ca(2+)](i) regulation and cell morphology.

Signaling in myocardial hypertrophy: life after calcineurin?

Stress pathways and heart failure.

A Ras-dependent pathway regulates RNA polymerase II phosphorylation in cardiac myocytes: implications for cardiac hypertrophy.

Cellular and functional defects in a mouse model of heart failure.

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