HDAC-mediated deacetylation of KLF5 associates with its proteasomal degradation.

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HDAC-mediated deacetylation of KLF5 associates with its proteasomal degradation.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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HDAC-mediated deacetylation of KLF5 associates with its proteasomal degradation.

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HDAC-mediated deacetylation of KLF5 associates with its proteasomal degradation.

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HDAC-mediated deacetylation of KLF5 associates with its proteasomal degradation.

@en

HDAC-mediated deacetylation of KLF5 associates with its proteasomal degradation.

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HDAC-mediated deacetylation of KLF5 associates with its proteasomal degradation.

@en

HDAC-mediated deacetylation of KLF5 associates with its proteasomal degradation.

@nl

P1476

HDAC-mediated deacetylation of KLF5 associates with its proteasomal degradation.

@en

P2093

Baotong Zhang

http://www.w3.org/2001/XMLSchema#string

Cara Rae Schiavon

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Jamie L King

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Jin-Tang Dong

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Ran Tao

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Ruoyu Tian

http://www.w3.org/2001/XMLSchema#string

Siyuan Xia

http://www.w3.org/2001/XMLSchema#string

Yixiang Li

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P2860

Genome engineering using the CRISPR-Cas9 system

Positive and negative regulation of the cardiovascular transcription factor KLF5 by p300 and the oncogenic regulator SET through interaction and acetylation on the DNA-binding domain

Human Kruppel-like factor 5 is a target of the E3 ubiquitin ligase WWP1 for proteolysis in epithelial cells

KLF5 inhibits angiogenesis in PTEN-deficient prostate cancer by attenuating AKT activation and subsequent HIF1α accumulation

HDAC2 phosphorylation-dependent Klf5 deacetylation and RARα acetylation induced by RAR agonist switch the transcription regulatory programs of p21 in VSMCs

The cyclin-dependent kinase inhibitor p21 is a crucial target for histone deacetylase 1 as a regulator of cellular proliferation.

The deacetylase HDAC1 negatively regulates the cardiovascular transcription factor Krüppel-like factor 5 through direct interaction.

Interruption of KLF5 acetylation converts its function from tumor suppressor to tumor promoter in prostate cancer cells.

The E3 ubiquitin ligase SMAD ubiquitination regulatory factor 2 negatively regulates Krüppel-like factor 5 protein.

Mifepristone Suppresses Basal Triple-Negative Breast Cancer Stem Cells by Down-regulating KLF5 Expression.

P31

scholarly article

P356

10.1016/J.BBRC.2018.04.153

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P407

English

P577

2018-04-18T00:00:00Z

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P698

29679567

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P932

5940529

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