Distinct molecular pathways mediate Mycn and Myc-regulated miR-17-92 microRNA action in Feingold syndrome mouse models.

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Distinct molecular pathways mediate Mycn and Myc-regulated miR-17-92 microRNA action in Feingold syndrome mouse models.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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Distinct molecular pathways me ...... eingold syndrome mouse models.

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Distinct molecular pathways me ...... eingold syndrome mouse models.

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Item

label

Distinct molecular pathways me ...... eingold syndrome mouse models.

@en

Distinct molecular pathways me ...... eingold syndrome mouse models.

@nl

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Distinct molecular pathways me ...... eingold syndrome mouse models.

@en

Distinct molecular pathways me ...... eingold syndrome mouse models.

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P1476

Distinct molecular pathways me ...... Feingold syndrome mouse models

@en

P2093

Anastasia Kozlova

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Elena Paltrinieri

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Fatemeh Mirzamohammadi

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Garyfallia Papaioannou

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Ugur M Ayturk

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P2860

Phosphorylation of the PTEN tail regulates protein stability and function

The tumor suppressor PTEN is phosphorylated by the protein kinase CK2 at its C terminus. Implications for PTEN stability to proteasome-mediated degradation

Germline deletion of the miR-17∼92 cluster causes skeletal and growth defects in humans

Targeted deletion reveals essential and overlapping functions of the miR-17 through 92 family of miRNA clusters

Lymphoproliferative disease and autoimmunity in mice with increased miR-17-92 expression in lymphocytes

miR-19 is a key oncogenic component of mir-17-92

The miR-17/92 cluster: a comprehensive update on its genomics, genetics, functions and increasingly important and numerous roles in health and disease

A microRNA polycistron as a potential human oncogene

c-Myc-regulated microRNAs modulate E2F1 expression

Feingold syndrome: clinical review and genetic mapping

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s41467-018-03788-7

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scholarly article

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10.1038/S41467-018-03788-7

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English

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Tatsuya Kobayashi

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2018-04-10T00:00:00Z

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29636449

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microRNA

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5893605

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