Gain-of-function mutations in granulocyte colony-stimulating factor receptor (CSF3R) reveal distinct mechanisms of CSF3R activation.

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Gain-of-function mutations in granulocyte colony-stimulating factor receptor (CSF3R) reveal distinct mechanisms of CSF3R activation.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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Gain-of-function mutations in ...... echanisms of CSF3R activation.

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Gain-of-function mutations in granulocyte colony-stimulating factor receptor

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Gain-of-function mutations in ...... echanisms of CSF3R activation.

@en

Gain-of-function mutations in granulocyte colony-stimulating factor receptor

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Gain-of-function mutations in ...... echanisms of CSF3R activation.

@en

Gain-of-function mutations in granulocyte colony-stimulating factor receptor

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P1476

Gain-of-function mutations in ...... echanisms of CSF3R activation.

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P2093

Cody Coblentz

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Haijiao Zhang

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Jasmine Means

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Jeffrey W Tyner

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Julia E Maxson

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Sophie Means

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P2860

Identification of a novel Stat3 recruitment and activation motif within the granulocyte colony-stimulating factor receptor

Oncogenic CSF3R mutations in chronic neutrophilic leukemia and atypical CML

Granulocyte colony-stimulating factor (G-CSF) treatment of childhood acute myeloid leukemias that overexpress the differentiation-defective G-CSF receptor isoform IV is associated with a higher incidence of relapse

Alternative modes of GM-CSF receptor activation revealed using activated mutants of the common beta-subunit.

Saturation mutagenesis of the beta subunit of the human granulocyte-macrophage colony-stimulating factor receptor shows clustering of constitutive mutations, activation of ERK MAP kinase and STAT pathways, and differential beta subunit tyrosine phos

Granulocyte colony-stimulating factor: molecular mechanisms of action during steady state and 'emergency' hematopoiesis.

Homodimeric cross-over structure of the human granulocyte colony-stimulating factor (GCSF) receptor signaling complex.

Escape from Cbl-mediated downregulation: a recurrent theme for oncogenic deregulation of receptor tyrosine kinases.

Granulocyte-macrophage colony-stimulating factor (GM-CSF) and granulocyte colony-stimulating factor (G-CSF): receptor biology, signal transduction, and neutrophil activation.

Csf3r mutations in mice confer a strong clonal HSC advantage via activation of Stat5.

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scholarly article

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10.1074/JBC.RA118.002417

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English

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Kevin Watanabe-Smith

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2018-03-23T00:00:00Z

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29572350

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P932

5950016

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