Dystrophin Expressing Chimeric (DEC) Human Cells Provide a Potential Therapy for Duchenne Muscular Dystrophy.

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Dystrophin Expressing Chimeric (DEC) Human Cells Provide a Potential Therapy for Duchenne Muscular Dystrophy.

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http://www.wikidata.org/entity/Q52652706

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

@zh-cn

2018年学术文章

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2018年学术文章

@zh-my

2018年学术文章

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2018年學術文章

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2018年學術文章

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name

Dystrophin Expressing Chimeric ...... r Duchenne Muscular Dystrophy.

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Dystrophin Expressing Chimeric

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type

Item

label

Dystrophin Expressing Chimeric ...... r Duchenne Muscular Dystrophy.

@en

Dystrophin Expressing Chimeric

@nl

prefLabel

Dystrophin Expressing Chimeric ...... r Duchenne Muscular Dystrophy.

@en

Dystrophin Expressing Chimeric

@nl

P1476

Dystrophin Expressing Chimeric ...... or Duchenne Muscular Dystrophy

@en

P2093

Ahlke Heydemann

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Enza Marchese

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Erzsebet Szilagyi

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Jesus Garcia

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Joanna Cwykiel

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Krzysztof Siemionow

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P2860

Local restoration of dystrophin expression with the morpholino oligomer AVI-4658 in Duchenne muscular dystrophy: a single-blind, placebo-controlled, dose-escalation, proof-of-concept study

Inflammation converts human mesoangioblasts into targets of alloreactive immune responses: implications for allogeneic cell therapy of DMD

Culture of human skeletal muscle myoblasts: timing appearance and localization of dystrophin-glycoprotein complex and vinculin-talin-integrin complex.

Quiescence of human muscle stem cells is favored by culture on natural biopolymeric films.

Long-term persistence of donor nuclei in a Duchenne muscular dystrophy patient receiving bone marrow transplantation.

A simplified immune suppression scheme leads to persistent micro-dystrophin expression in Duchenne muscular dystrophy dogs

Immunomodulatory Effects of Different Cellular Therapies of Bone Marrow Origin on Chimerism Induction and Maintenance Across MHC Barriers in a Face Allotransplantation Model.

Postnatal genome editing partially restores dystrophin expression in a mouse model of muscular dystrophy.

Dystrophin expression in muscle stem cells regulates their polarity and asymmetric division.

Nuclear positioning in muscle development and disease.

P2888

s12015-018-9807-z

P304

370-384

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scholarly article

P356

10.1007/S12015-018-9807-Z

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P433

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P478

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Maria Siemionow

P577

2018-06-01T00:00:00Z

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P698

29546607

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P932

5960489

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