Enhanced cyclooxygenase-2 activity leads to intestinal dysmotility following hemorrhagic shock.

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Cyclooxygenase‐2 facilitates dengue virus replication and serves as a potential target for developing antiviral agents Immediate postoperative evaluation of enteric anastomosis after the use of meloxicam in rats

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Enhanced cyclooxygenase-2 activity leads to intestinal dysmotility following hemorrhagic shock.

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Enhanced cyclooxygenase-2 acti ...... y following hemorrhagic shock.

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Enhanced cyclooxygenase-2 acti ...... y following hemorrhagic shock.

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Enhanced cyclooxygenase-2 acti ...... y following hemorrhagic shock.

@en

Enhanced cyclooxygenase-2 acti ...... y following hemorrhagic shock.

@nl

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Enhanced cyclooxygenase-2 acti ...... y following hemorrhagic shock.

@en

Enhanced cyclooxygenase-2 acti ...... y following hemorrhagic shock.

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Enhanced cyclooxygenase-2 acti ...... y following hemorrhagic shock.

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Li-Min Zhang

http://www.w3.org/2001/XMLSchema#string

Xiu-Qing Hao

http://www.w3.org/2001/XMLSchema#string

Ya-Ting Tian

http://www.w3.org/2001/XMLSchema#string

Yu-Ping Zhang

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Pathophysiology of motility dysfunction in bowel obstruction: role of stretch-induced COX-2

Mechano-transcription of COX-2 is a common response to lumen dilation of the rat gastrointestinal tract.

Cyclooxygenase-2 deficiency leads to intestinal barrier dysfunction and increased mortality during polymicrobial sepsis.

COX-2 dependent inflammation increases spinal Fos expression during rodent postoperative ileus.

EP2 and EP4 receptors on muscularis resident macrophages mediate LPS-induced intestinal dysmotility via iNOS upregulation through cAMP/ERK signals.

Early trauma-hemorrhage-induced splenic and thymic apoptosis is gut-mediated and toll-like receptor 4-dependent.

COX-1 (PTGS1) and COX-2 (PTGS2) polymorphisms, NSAID interactions, and risk of colon and rectal cancers in two independent populations.

Regulation of smooth muscle contraction by the epithelium: role of prostaglandins.

Cox-2 is regulated by Toll-like receptor-4 (TLR4) signaling: Role in proliferation and apoptosis in the intestine.

Oxidative modification of the intestinal mucus layer is a critical but unrecognized component of trauma hemorrhagic shock-induced gut barrier failure.

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838-843

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scholarly article

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10.1590/S0102-865020150120000008

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2015-12-01T00:00:00Z

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Enhanced cyclooxygenase-2 activity leads to intestinal dysmotility following hemorrhagic shock.

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Enhanced cyclooxygenase-2 activity leads to intestinal dysmotility following hemorrhagic shock.

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