The tobacco carcinogen NNK drives accumulation of DNMT1 at the GR promoter thereby reducing GR expression in untransformed lung fibroblasts.

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The tobacco carcinogen NNK drives accumulation of DNMT1 at the GR promoter thereby reducing GR expression in untransformed lung fibroblasts.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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The tobacco carcinogen NNK dri ...... ntransformed lung fibroblasts.

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The tobacco carcinogen NNK dri ...... ntransformed lung fibroblasts.

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The tobacco carcinogen NNK dri ...... ntransformed lung fibroblasts.

@en

The tobacco carcinogen NNK dri ...... ntransformed lung fibroblasts.

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The tobacco carcinogen NNK dri ...... ntransformed lung fibroblasts.

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The tobacco carcinogen NNK dri ...... ntransformed lung fibroblasts.

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P1476

The tobacco carcinogen NNK dri ...... ntransformed lung fibroblasts.

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P2093

Kerryn M Taylor

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Nimisha Singh

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Roxanne Wheeler

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P2860

Accurate normalization of real-time quantitative RT-PCR data by geometric averaging of multiple internal control genes

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The human glucocorticoid receptor: one gene, multiple proteins and diverse responses

DNA methylation, methyltransferases, and cancer.

The tobacco-specific carcinogen NNK induces DNA methyltransferase 1 accumulation and tumor suppressor gene hypermethylation in mice and lung cancer patients.

Glucocorticoid receptor over-expression promotes human small cell lung cancer apoptosis in vivo and thereby slows tumor growth.

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scholarly article

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10.1038/S41598-018-23309-2

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English

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Dalene Vosloo

David W. Ray

Paula Sommer

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2018-03-20T00:00:00Z

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29559689

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5861118

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