Glycogen synthase kinase 3β inhibition reduces mitochondrial oxidative stress in chronic myocardial ischemia.

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Glycogen synthase kinase 3β inhibition reduces mitochondrial oxidative stress in chronic myocardial ischemia.

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2018 nî lūn-bûn

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Glycogen synthase kinase 3β in ...... n chronic myocardial ischemia.

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Glycogen synthase kinase 3β in ...... n chronic myocardial ischemia.

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Glycogen synthase kinase 3β in ...... n chronic myocardial ischemia.

@en

Glycogen synthase kinase 3β in ...... n chronic myocardial ischemia.

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Glycogen synthase kinase 3β in ...... n chronic myocardial ischemia.

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Glycogen synthase kinase 3β in ...... n chronic myocardial ischemia.

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Glycogen synthase kinase 3β in ...... n chronic myocardial ischemia.

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Brittany A Potz

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Frank W Sellke

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Laura A Scrimgeour

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Neel R Sodha

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Richard T Clements

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Sharif A Sabe

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P2860

Glycogen synthase kinase-3 regulates mitochondrial outer membrane permeabilization and apoptosis by destabilization of MCL-1

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GSK3: a multifaceted kinase in Wnt signaling.

Glycogen synthase kinase-3beta regulates post-myocardial infarction remodeling and stress-induced cardiomyocyte proliferation in vivo

Serine 332 phosphorylation of insulin receptor substrate-1 by glycogen synthase kinase-3 attenuates insulin signaling.

Cdc42-mediated inhibition of GSK-3β improves angio-architecture and lumen formation during VEGF-driven pathological angiogenesis.

What Are the bona fide GSK3 Substrates?

Calpain inhibition decreases myocardial apoptosis in a swine model of chronic myocardial ischemia.

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scholarly article

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10.1016/J.JTCVS.2017.12.127

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English

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2018-02-02T00:00:00Z

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29523407

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5960424

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