Chronic kidney disease induces a systemic microangiopathy, tissue hypoxia and dysfunctional angiogenesis.

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Chronic kidney disease induces a systemic microangiopathy, tissue hypoxia and dysfunctional angiogenesis.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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Chronic kidney disease induces ...... nd dysfunctional angiogenesis.

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Chronic kidney disease induces ...... nd dysfunctional angiogenesis.

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label

Chronic kidney disease induces ...... nd dysfunctional angiogenesis.

@en

Chronic kidney disease induces ...... nd dysfunctional angiogenesis.

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Chronic kidney disease induces ...... nd dysfunctional angiogenesis.

@en

Chronic kidney disease induces ...... nd dysfunctional angiogenesis.

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Chronic kidney disease induces ...... and dysfunctional angiogenesis

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Bettina Reglin

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Hamoud Nasser

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Hans-Ulrich Prommer

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Johannes Maurer

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Karoline von Websky

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Kerstin Sommer

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Rudi Samapati

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Uwe Querfeld

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Hypoxia promotes fibrogenesis in vivo via HIF-1 stimulation of epithelial-to-mesenchymal transition

Endothelial to Mesenchymal Transition (EndoMT) in the Pathogenesis of Human Fibrotic Diseases

Hypoxia: The Force that Drives Chronic Kidney Disease

Quantitative Micro-Computed Tomography Imaging of Vascular Dysfunction in Progressive Kidney Diseases

Uremic myopathy: is oxidative stress implicated in muscle dysfunction in uremia?

Angiotensin II overcomes strain-dependent resistance of rapid CKD progression in a new remnant kidney mouse model

Responses of the skin microcirculation to acetylcholine and to sodium nitroprusside in chronic uremic patients.

The endothelial surface layer.

Increased concentration of circulating angiogenesis and nitric oxide inhibitors induces endothelial to mesenchymal transition and myocardial fibrosis in patients with chronic kidney disease.

Stable expression of HIF-1alpha in tubular epithelial cells promotes interstitial fibrosis

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5317

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scholarly article

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10.1038/S41598-018-23663-1

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English

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Christian Freise

Pedro Guimarães

Axel R Pries

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2018-03-28T00:00:00Z

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29593228

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5871820

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